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NK细胞及IL-12、IFN-γ参与多发性硬化的免疫病理过程(综述) 被引量：3

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摘要 综述了近年来自然杀伤细胞、白细胞介素 -1 2和干扰素 -γ参与多发性硬化的免疫发病机制及免疫病理过程的研究进展。
作者 闫晓波 韩葳 王维治 王化冰
机构地区 哈尔滨医科大学附属第二医院神经科
出处 《中国神经免疫学和神经病学杂志》 CAS 2001年第4期225-227,共3页 Chinese Journal of Neuroimmunology and Neurology
关键词 多发性硬化 自然杀伤细胞 白细胞介素-12 干扰素-Γ 病理
分类号 R744.510.2 [医药卫生—神经病学与精神病学]
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参考文献10

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  • 2Macatonia SE,Hosken NA,Litton M,et al.Dendritic cells produce IL-12 and direct the development of Th1 cells from naive CD4+ T cells[].J Immunol.1995
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  • 4Wang X,Chen M,Wandinger KP,et al.IFN-beta1b inhibits IL-12 production in peripheral blood mononuclear cells in an IL-10-dependent mechanism:relevance to IFN-beta-1b therapeutic effects in multiple sclerosis[].J Immunol.2000
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同被引文献29

  • 1许明智,杨玲玲.细胞因子的神经生物学效应[J].国外医学（精神病学分册）,1997,24(1):1-5. 被引量：21
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  • 6[6]Maes M, Meltzer H, Bosmans E, et al. Increased plasma concentrations of interleukin-6, soluble interleukin-6 receptor, soluble interleukin-2 receptor and transferrin receptor in major depression. J Affect Disord,1995,34:301～309
  • 7Naudin J, Capo C, Giusano B, et al. A differential role for interleukin-6 and tumor necrosis factor-α in Schizophrenia. Schizophr Res, 1997, 2-3(26): 227.
  • 8Frommberger UH, Bauer J, Haselbauer P, et al. Interleukin-6 (IL-6)plasma levels in depression and schizophrenia: Comparison between the acute state and after remission. Eur Arch Psychiatry Clin Neurosci,1997, 4(247): 228.
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  • 10Cazzullo CL, Saechetti E, Galluzzo A, et al. Cytokine profiles in drugnive schizophreie patients. Schizophr Res, 2001, 2- 3(47): 293.

引证文献3

二级引证文献18

中国神经免疫学和神经病学杂志
2001年 第4期

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