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乌司他丁对急性肺损伤水通道蛋白-1调节机制的研究 被引量：3

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摘要 乌司他丁治疗急性肺损伤（ALI）的机制是否与水通道蛋白（AQPs）的表达有关，乌司他丁调节水通道蛋白的机制是否是通过抑制炎症介质来实现，对此，我们进行了研究．现报告如下。
作者 王锐 刘虹
机构地区 山西医科大学第一医院ICU
出处 《中国药物与临床》 CAS 2009年第4期315-317,共3页 Chinese Remedies & Clinics
关键词 水通道蛋白-1 急性肺损伤 乌司他丁 调节 炎症介质
分类号 R96 [医药卫生—药理学]
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共引文献6

同被引文献26

  • 1彭洁,张虹,李涛,李中国,吴云霞.Effect of Dexamethasone and Aquaporin-1 Antisense Oligonucleotides on the Aquaporin-1 Expression in Cultured Human Trabecular Meshwork Cells[J].Journal of Huazhong University of Science and Technology(Medical Sciences),2006,26(1):137-140. 被引量：7
  • 2游志坚,姚尚龙,梁华根.不同时间单肺通气后兔两侧肺损伤程度比较[J].中国急救医学,2007,27(2):133-135. 被引量：44
  • 3张伟,谢灿茂,黄建强,廖槐,柯彩霞.高渗液对大鼠胸膜间皮细胞水通道蛋白1表达的影响[J].中华医学杂志,2007,87(35):2508-2511. 被引量：4
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  • 10Towne JE, Krane CM, Bachurski CJ, et al. Tumor necrosis factor-alpha inhibits aquaporin 5 expression in mouse lung ep- itheliaI cells. J Biol Chem, 2001,276 (22) : 18657 18664.

引证文献3

二级引证文献19

中国药物与临床
2009年 第4期

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