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应激性高血糖的原因及发生机制 被引量:12

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作者 敦社林 李伟
出处 《河北医药》 CAS 2008年第7期1036-1038,共3页 Hebei Medical Journal
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参考文献19

  • 1Mesotten D, van den Berghe G. Clinical potential of insulin therapy in critically ill patients. Drugs, 2003,63 : 625-636.
  • 2Lewis KS, Kane-Gill SL, Bobek MB, et al. Intensive insulin therapy for critically ill patients. Ann Pharmacother,2004,38:1243-1251.
  • 3Orford NR. Intensive insulin therapy in septic shock . Crit Care Resusc, 2006,8 : 230-234.
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  • 5Robinson LE, van Soeren MH. Insulin resistance and hyperglycemia in critical illness: role of insulin in glycemic control. AACN Clin Issues,2004, 15:45- 62.
  • 6Marik PE, Raghavan M. Stress-hyperglycemia, insulin and immunomodulation in sepsis. Intensive Care Meal,2004,30: 748-756.
  • 7Dunger D Yuen K, Ong K. Insulin-like growth factor I and impaired glucose tolerance. Horm Res,2004,62: 101-107.
  • 8Montori VM, Bistfian BR, McMahon MM. Hyperglycemia in acutely ill patients. JAMA,2002,288:2167-2169.
  • 9Waclllin G, Augstein P, Schroder D, et al. IL-lbeta, IFN-gamma and TNF- alpha increase vulnerabihtv of pancreatic beta cells to autoimmtme destruction. J Autoimmun, 2003,20:303-312.
  • 10withers D J, Gutierrez JS, Towery H. et al. Disruption of IRS-2 causes type 2 diabetes in mice. Nature, 1998,391 : 900-904.

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