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高浓度氧肺损伤机制研究进展 被引量:5

The advance of research on the mechanism of hyperoxia-induced lang injury
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出处 《实用儿科临床杂志》 CAS CSCD 北大核心 2003年第12期998-1000,共3页 Journal of Applied Clinical Pediatrics
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参考文献15

  • 1[1]Singer L, Yamashita T, Lilien L,et al.A longitudinal study of developmental outcome of infants with bronchopulmonary dysplasia and very low birth weight[J].Pediatrics,1997,100(6): 987~993.
  • 2[2]Manji JS, O′Kelly CJ, Leung WI,et al.Timing of hyperoxic exposure during alveolarization influences damage mediated by leukotrienes[J]. Am J Physiol Lung Cell Mol Physiol, 2001,281:L799~L806.
  • 3[3]Horinouchi H, Wang CC, Shepherd KE,et al. TNF alpha gene and protein expression in alveolar macrophages in acute and chronic hyperoxia-induced lung injury[J]. Am J Respir Cell Mol Biol, 1996,14(6):548~555.
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  • 7[7]Johnston CJ, Stripp BR, Piedbeouf B,et al.Inflammatory and epithelial responses in mouse strains that differ in sensitivity to hyperoxic injury[J]. Exp Lung Res,1998,24(2):189~202.
  • 8[8]Piedboeuf B, Gamache M, Frenette J,et al.Increased endothelial cell expression of platelet-endothelial cell adhesion molecule-1 during hyperoxic lung injury[J]. Am J Respir Cell Mol Biol,1998,19(4):543~553.
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  • 10[10]Buckley S, Barsky L, Driscoll B,et al.Apoptosis and DNA damage in type 2 alveolar epithelial cells cultured from hyperoxic rats[J]. Am J Physiol,1998,274(5 Pt 1):L714~L720.

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