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急性心肌梗死后心室重塑——心肌细胞凋亡的意义 被引量:16

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出处 《中华心血管病杂志》 CAS CSCD 北大核心 2003年第8期629-631,共3页 Chinese Journal of Cardiology
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  • 1Cheng W, Kajstura J, Nitahara JA, et al. Programmed myocyte cell death affects the viable myocardium after infarction in rats. Exp Cell Res, 1996,226:316-327.
  • 2Bialik S, Geenen DL, Sassen IE, et al. Myocyte apoptosis during acute myocardial infarction in the mouse localizes to hypoxic regions but occurs independently of p53. J Clin Invest, 1997,100: 1363-1372.
  • 3Li WG, Zaheer A, Coppey L, et al. Activation of JNK in the remote myocardium after large myocardial infarction in rats Biochemi Biophysi Res Commu, 1998, 246:816-820.
  • 4Sam F, Sawyer DB, Chang DL, et al. Progressive left ventricular remodeling and apoptosis late after myocardial infarction on mouse heart. Am J Physiol Heart Cire Physiol, 2000,279: H422-H428.
  • 5Olivetti G, Quaini F, Sala R, et al. Acute myocardial infarction in humans is associated with activation of progranned myocyte cell death in the surviving portion of the heart. J Mol Cell Cardiol, 1996,28: 2005-2016.
  • 6Li Q, Li B, Wang X, et al. Overexpression of insulin-like growth factor-1 in mice protects from myocyte death after infarction, attenuating ventricular dilation, wall stress, and cardiac hypertrophy. J Clin Invest, 1997,100 : 1991-1999.
  • 7Teiger E, Dam TV, Richard L, et al. Apoptosis in pressure overload-induced heart hypertrophy in the rat. J Clin Invest, 1996, 97 : 2891-2897.
  • 8Sam F, Sawyer DB, Xie Z, et al. Mice lacking inducible nitric oxide synthase have improved left ventricular contractile function and reduced apoptotic cell death late after myocardial infarction. Circ Res, 2001,89:351-356.
  • 9Anversa P, Cheng W, Liu Y, et al. Apoptosis and myocardial infarction. Basic Res Cardiol, 1998,93 : [ Suppl] 3,8-12.
  • 10Krown KA, Page MT, Nguyen C, et al. Tumor necrosis factor alpha-induced apoptosis in cardiac myocytes : Involement of the sphingolipid signaling cascade in cardiac cell death. J Clin Invest, 1996, 98:2854-2865.

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