摘要
目的 :研究孤束核 臂旁核 中央杏仁核间接纤维联系方式 ,并对臂旁核内一氧化氮的作用途径进行了探讨。方法 :利用直流电损毁孤束核顺行溃变 ,结合中央杏仁核注射辣根过氧化物酶逆行追踪技术 ,在超微结构水平上 ,观察大鼠臂旁核内溃变终末及逆行标记结构的形态分布及其相互关系。结果 :分布于臂旁核内孤束核的轴突终末 ,可与投射至中央杏仁核的神经元及其他结构形成十分密切的并列关系。结论 :臂旁核内的突触并列结构可能是一氧化氮发挥有效生物信使与信息分子作用的结构基础。
Objective:To study the connection pathway of the solitary tract mucleus the parabrachial nucleus the central amygdaloid mucleus ,and to explore the role of the nitric oxide. Medithods:Based on anterograde degeneration caused by electolytic lesions in the solitray tract nucleus and HRP retrograde trcing techniques by injection in the central amygdaloid nucleus, We observe degeneration terminals and retrograde tracing structures in the parabrachial nucleus under electron microscope. Results:In the parabrachial nucleus, the solitary tract nucleus terminals were close to other neuronal structures,including the neurons projecting to the central amyadaloid nuclesu. Conclusion: In the parabrachial nucleus,the synaptic apposition may be the basic mechanism for NO as biological messenger and signal molecule.
出处
《解剖学杂志》
CAS
CSCD
北大核心
2002年第4期369-374,共6页
Chinese Journal of Anatomy
基金
江苏省卫生厅青年基金项目 (Q6 5 0 4)
南京医科大学科技发展基金项目 (NY990 0 6 )
关键词
孤束核
臂旁核
中央杏仁核
大鼠
超微结构
parabrachial nucleus
solitary tract nucleus
central amygdaloid nucleus
rat
