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加锌停跳液心肌保护的动物实验研究 被引量:1

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摘要 本实验显示锌在心肌缺血、再灌注中,既能阻止氧自由基产生,加强清除自由基能力,稳定心肌细胞膜,又可作为Ca^(11)拮抗剂和抗氧化物质,最终减少了心肌摄Ca^(11)并降低心肌张力,加强对ATP的保护,有利于心功能的恢复。本实验结果初步显示,冷钾停跳液加锌可有效地预防或减轻长时间心脏缺血及再灌注损伤,是心肌保护课题中值得进一步研究较为理想的物质。
出处 《心肺血管学报》 1991年第1期36-38,共3页
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