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进行性肾损害的可能机制——慢性缺氧学说 被引量:4

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作者 饶向荣 李深
出处 《中国中西医结合肾病杂志》 2001年第6期370-372,共3页 Chinese Journal of Integrated Traditional and Western Nephrology
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  • 1Fine LG, Orphanides C, Norman JT.Progressive renal disease: The chronic hypothesis. Kidney Int, 1998,53: 74-78.
  • 2Anderson S, Brenner M. Progressive renal diseases: a disorder of adaptation. Q J Med, 1989,70:185-189.
  • 3Remuzzi G, Ruggenti P, Benigin A. Understanding the nature of renal diseases progression. Kidney Int, 1997, 51:12-15.
  • 4The GISEN Group: Randomised placebo-controlled trail of the effect of ramipril on decline glomerular filtration rate and risk of terminal renal failure in proteinuric, non diabetic nephropathy. Lancet, 1997, 349:1887-1863.
  • 5Fine LG, Bandyopadhay, Norman JT. Is there a common mechanism for the progression of different types of renal disease other than proteinuria? Towards the unifying theme of chronic hypoxia. Kidney Int, 2000, 57: 22-26.
  • 6Bauer C, Kurtz A: Oxygen sensing on the cellular and molecular levels. Kindney Int, 1997, 51:74-78.
  • 7Bunn HF, Poyton R. Oxygen senting and molecular adaptation to hypoxia. Physiol Rew, 1996,76: 839-885.
  • 8hih S-C, Claffey KP. Hypoxia regulation of gene expression in mammalian cells. Iht J Exp Pathol, 1998,70: 374-357.
  • 9Semenza GL, agani F, Booth G, et al. Structural and functional analysis of hypoxia-inducible factor 1.Kindney Int, 1997, 51: 553-555.
  • 10Jill T, Norman, Ian M, et al. Garcia: Hypoxia promotes fibrogenesis in human renal fibroblasts. Kidney Int, 2000,58:2351-2366.

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