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抑制炎症的细胞因子与肾小球肾炎的关系 被引量:2

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摘要 在肾小球肾炎发病机理研究中 ,细胞因子网络调节作用在肾小球肾炎发生发展及肾小球硬化中发挥重要作用。致炎性与抗炎性细胞因子相互作用维持机体免疫系统的平衡。IL 1及TNF是由炎症细胞产生的主要前炎症细胞因子。主要由Th2细胞分泌的抗炎性细胞因子如IL 4、IL 1 0、IL 1 3也能被用于抑制IL 1及TNF合成和增加其拮抗剂产生。抗炎性细胞因子 ,主要是IL 4、IL 1 0、IL 1 3、IL 6与肾脏炎症疾病关系研究亦有一定进展。
出处 《国外医学(泌尿系统分册)》 2001年第4期164-165,共2页 Foreign Medical Sciences(Urology and Nephrology Foreign Medical Sciences)
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  • 1谵贻璞,余学清.肾内科学[M].北京:人民卫生出版社,2008:23.
  • 2[1]Tilg H,Trehu E,Atkins MB,et al.interleukin-6(IL-6) as an anti-inflammatory cytokine:induction of circulating IL-1 receptor antagonist and soluble tumor necrosis factor receptor p55[ J ].Blood,1994,83 (1):113-118.
  • 3[3]Floege J,Eng E,Young BA,et al.Infusion of platelet-derived growth factor or basic fibroblast growth factor induces selective glomerular mesangial cell proliferation and matrix accumulation in rats[ J].J Clin Invest,1993,92(6):2952-2962.
  • 4[5]Nakamura T,Ebihara I,Takahashi T,et al.Increased interleukin 6 mRNA expression by peripheral blood T cells from patients with IgA nephropathy[ J].Autoimmunity,1993,15(3):171-179.
  • 5[6]Harada K,Akai Y,Kurumatani N,et al.Prognostic value of urinary interleukin 6 in patients with IgA nephropathy:an 8-year follow-up study[J \ ].Nephron.,2002.92(4):824-826.
  • 6[7]Syrjanen J,Hurme M,Lehtimaki T.et al.Polymorphism of the cytokine genes and IgA nephropathy[ J].Kidney-Int.,2002,61 (3):1079-1085.
  • 7[8]Maksic D,Spasic P,Dimitrijevic J et al.,Significance of inflammatory cytokines in the pathogenesis of IgA nephropathy[J].Vojnosanit Pregl,1998,55(2):141-149.
  • 8[9]Taniguchi Y,Yorioka N,Kumagai J.et al.Interleukin-6 localization and the prognosis of IgA nephropathy[J].Nephron,1999,81(1):94-98.
  • 9[10]Baba Y,Akagi H,Fukushima K.et al.Quantitative analysis of interleukin 6 (IL-6) in patients with IgA nephropathy after tonsillectomy[J].Auris Nasus Larynx,1999,26(2):177-182.
  • 10[11]Kaneko K,Nagaoka R,Ohtomo Y,et al.Effect of mizoribine on IL-6release by peripheral blood mononuclear cells[J].Nephron,2002,91(3):506-8.

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