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Bax基因过表达诱导肝癌细胞凋亡 被引量:2

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出处 《中华消化杂志》 CAS CSCD 北大核心 2001年第2期114-116,共3页 Chinese Journal of Digestion
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  • 5Yang E, Zha J, Jockel J, et al. Bad, a heterodimeric partner forbcl-xl and bcl-2, displace bax and promotes cell death[J]. Cell, 1995, 80 (2) :285-291.
  • 6Oltvai ZN, Milliman CL, Korsmeyer SJ. bcl-2 heterodimerizes in vivo with a conserved homolog, bax, that accelerates programmed cell death[J]. Cell, 1993, 74(4): 609-619.
  • 7Nor JE, Christensen J, Liu J, et al. Up-Regulation of bcl-2 in microvascular endothelial cells enhances intratumoral angiogenesis and accelerates tumor growth [J]. Cancer Res, 2001,61 (5): 2183-2188.
  • 8Pidgeon GP, Barr MP, Harmey JH, et al. Vascular endotheli al growth factor (VEGF) upregulates BCL-2 and inhibits apoptosis in human and murine mammary adenocarcinoma cells [J]. Br J Cancer, 2001, 85(2): 273-278.Takehara T, Liu X, Fujimoto J, et al. Expression and role of bcl-xl in human hepatocellular carcinomas[J]. Hepatology, 2001, 34 (1): 55-61.
  • 9顾炜,彭志海,裘国强,张益,王兆文.肝癌组织中凋亡信号蛋白的表达及其临床意义[J].中国肿瘤,2003,12(1):48-50. 被引量:11
  • 10陆东东.肝癌中iNOS与P16、Bax的表达及其与细胞凋亡关系[J].临床肝胆病杂志,2003,19(1):58-59. 被引量:4

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