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小干扰RNA与口腔癌研究进展综述 被引量:1

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摘要 口腔癌是口腔外科常见恶性肿瘤,近年来的研究显示其发生、发展、侵袭转移和预后与体内小干扰RNA表达水平密切相关。因此本文通过对小干扰RNA产生机制、与肿瘤的发生、与口腔癌前病变、与口腔癌及与口腔癌的治疗等方面综述如下。
作者 吴国聪
出处 《河南外科学杂志》 2013年第3期62-63,共2页 Henan Journal of Surgery
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  • 1van KM,Kedde M,Agami R.MicroRNA regulation by RNA-binding proteins and its implications for cancer[J].Nat Rev Cancer,2011,11(9):644-656.
  • 2Lu YC,Chen YJ,Wang HM,et al.Oncogenic function and early detection potential of miRNA-10b in oral cancer asiden-tified by microRNA profiling[J].Cancer Prev Res(Phila).2012.5(4):665-74.
  • 3Shi Z,Johnson JJ,Stack MS.Fluorescence In Situ Hybridiza-tion for MicroRNA Detection in Archived Oral CancerTissues[J].J Oncol,2012,20(12):e903581.
  • 4Yan B,Fu Q,Lai L,et al.Downregulation of microRNA99a in oral squamous cell carcinomas contributes tothe growth and survival of oral cancer cells[J].Mol Med Report.2012.6(3):675-681.
  • 5Chu YH,Tzeng SL,Lin CW,et al.Impacts of microRNA gene polymorphisms on the susceptibility of environmentalfac-tors leading to carcinogenesis in oral cancer[J].PLOS ONE.2012,7(6):e39777.
  • 6Rather MI,Nagashri MN,Swamy SS,et al.Oncogenic mi-croRNA-155downregulates tumor suppressor CDC73and pro-motes oralsquamous cell carcinoma cell proliferation:Implica-tions for cancer therapeutics[J].J Biol Chem,2012,8(2):312-317.
  • 7Jakymiw A,Patel RS,Deming N,et al.Overexpression of dic-er as a result of reduced let-7MicroRNA levels contributes to increased cell proliferation of oral cancer cells[J].Genes Chromosomes Cancer,2010,49(6):549-559.
  • 8Uesugi A,Kozaki K,Tsuruta T,et al.The tumor suppressive microRNA miR-218targets the mTOR component Rictor and-inhibits AKT phosphorylation in oral cancer[J].Cancer Res,2011,71(17):5765-578.
  • 9Kim JS,Yu SK,Lee MH,et al.MicroRNA-205directly reg-ulates the tumor suppressor,interleukin-24,in human KBoral cancer cells[J].Mol Cells,2012,12(3):414-419.
  • 10Catto JW,Alcaraz A,Bjartell AS,et al.MicroRNA in pros-tate,bladder,and kidney cancer:a systematic review[J].Eur Urol,2011,59(5):671-681.

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