1[1]Mahon N G, O'Roke, Codd M B, et al. Hospital mortality of acute myocardial infarction in the thrombolytic era. Heart, 1999, 81:478-482.
2[2]Cohn J N, Roberto F, Norman S, et al. Cardiac remodeling-concepte and clinical implications:A consensus paper from an international forum on cardiac remodeling. J Am Coll Cardiol, 2000, 35:569-582.
3[3]Givertz M M, Collucci W S. New targets for heart-failure therapy: Endothelin ,inflammatory cytokines, oxidative stress. Lancet, 1998, 352(Suppl I):34-38.
4[4]Yousef Z R, Redwood S R, Marber M S. Postinfarction left ventricular remodeling:Where are the theories and trials leading us? Heart, 2000, 83:76-80.
5[5]Vantrimpont P, Rouleau J L, Ciampi A, et al. Two-year course and significance of neurohumoral activation in the survival and ventricular enlargement (SAVE) study. Eur Heart J, 1998, 19:1552-1563.
6[6]Scheuer J. Catecholamines in cardiac hypertrophy. Am J Cardiol, 1999, 83:70-74H.
7[7]Collucci W S. Molecular and cellular mechanisms of myocardial failure. Am J Cardiol, 1997, 80: 15L-25L.
8[8]Olivetti G, Abbi R, Quaini F, et al. Apoptosis in the failing human heart. N Engl J Med, 1997, 336:1131-1141.
9[9]Aleksandar D, Aleksandar N, Jelera, et al. Acute and long term effect of thrombolysis after anterior wall acute myocardial infarction with serial assessment of infarct expansion and late ventricular remodeling. Am J Cardiol, 1996,77:446-452.
10[10]Marber M S, Taylor D J,Kloner R A. The open artery hypothesis: To open, or not to open, that is the question. Eur Heart J, 1996, 17:505-509.
