1Rice C, Birnbaum LS, Cogliano J. et al. Exposure assessment for endocrine disruptors:some considerations in the design of studies [J]. Environ Health Perspect, 2003, 111 (13): 1683-1690.
2Singleton DW, Khan SA. X enoestrogen exposure and mechanisms of endocrine disruptions[J]. Front Biosci, 2003,8 (S) : 110- 118.
3Colborn T,vom Saal FS, Soto AM. Developmental effects of endocrine-disrupting chemicals in wildlife and humans[J]. Environ Health Perspect, 1993,101 (5) : 378-384.
4Recknor JG, Reigart JR, Darden PM, et al. Prenatal care and infant lead exposure[J]. J Pediatr, 1997,130 (1):123- 127.
5Gonzalez CT, Peterson KE, Sanin LH, et al, Decrease in birth wight in relation to maternal bone lead burden[J]. Pediatrics, 1997,100(5) : 856-862.
10Gray LE Jr, Ostby J, Cooper RL, et al. The estrogenic and antiand rogenic pesticide methoxychlor alters the reproductive tract and behavior without affecting pituitary size or LH and prolactin secretion in male rats [J]. Toxicol Ind Health, 1999,15(1-2) :37-47.
5Staples CA, Dorn PB, Klecka GM, et al. A review of the environmentalfate, effects, and exposures of bisphenol A. Chemosphere,1998,36(10):2149-2173
6Olea N, Pulgar R, Perez P, et al. Estrogencity of resin-based compositesand senlants used in dentistry. Environ Health Perspect, 1996, 104: 298-305
7Jolanki R, Kanerva L, Estlander T. Occupational allergic contactdermatitis caused by epoxy diacrylate in ultraviolet-light-cured paint, and bisphenol A indental composite resin. Contact Dermatitis, 1995,33 (2): 94-99
8Hardin BD, Bond GP, Sikov MR, et al. Testing of select workplace chemicals for teratogenic potential. Scand J Work Environ.Health,1981,7(suppl.4):66-75
9Morrisey RE, George JD, Price CJ, et al. The developmental toxicity of bisphenol A in rats and mice. Fundam Appl Toxicol, 1987,8:571-582
10New DA, Cockroft DL. A rotating bottle culture method with continuous replacement of the gas phase. Experientia,1979, 35(1): 138-140
