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MR磁敏感加权成像在脑梗死治疗前后随访中的应用价值 被引量:3

Clinical Values of MR Susceptibility Weighted Imaging in the Follow-up of Cerebral Infarction Before and After Treatment
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摘要 目的探讨MR磁敏感加权成像(SWI)在脑梗死治疗前后随访中的应用价值。资料与方法搜集经临床及影像学检查证实的脑梗死患者资料30例,急性13例,亚急性17例。所有患者治疗前后均行常规MR平扫、扩散加权成像(DWI)及SWI扫描。对比分析治疗前后MRI图像变化特点。结果治疗前常规MRI检出脑梗死灶内出血4例,SWI检出9例,治疗后SWI显示5例发生出血性转化。治疗前30例脑梗死灶SWI图像相对信号强度为0.97±0.09,治疗后相对信号强度为1.06±0.12,治疗前后相对信号强度差异有统计学意义(t=-3.579,P<0.05)。治疗前后脑梗死灶内血管的显示差异有统计学意义(P<0.05)。结论SWI对于显示脑梗死伴发出血、治疗后出血性转化、侧支血管构建等方面优于常规MRI序列,在脑梗死治疗后随访中具有重要的临床应用价值。 Objective To investigate the value of MR susceptibility weighted imaging (SWI) in the follow-up of cerebral infarction before and after treatment. Materials and Methods 30 patients with cerebral infarction (acute infarction in 13 and subacute infarction in 17 patients) proved by clinical or imaging data were enrolled in this study including. All cases underwent conventional plain MRI scan,DWI and SWI. The changes of infarction before and after treatment were compared. Results Before treatment,hemorrhage within infarction was detected in 4 patients by routine MRI sequences,meanwhile in 9 patients by SWI. Post-treatment SWI revealed hemorrhagic transformation in 5 patients. The relative signal intensity of 30 lesions was 0.97±0.09 on SWI before treatment,while 1.06±0.12 after treatment. The difference of relative signal intensity has statistical significance(t=-3.579,P0.05)before and after treatment. The visualization rate of vessels inside infarction observed by SWI has statistical significance(P0.05)before and after treatment. Conclu-sion SWI is much better than routine MRI sequences in demonstrating hemorrhage,hemorrhagic transformation of infarction and blood vessels reconstruction after treatment,and has important clinical value in the follow-up of cerebral infarction.
出处 《临床放射学杂志》 CSCD 北大核心 2010年第5期574-578,共5页 Journal of Clinical Radiology
基金 重庆医科大学校级科研课题(XBYB2007038)
关键词 磁敏感加权成像 磁共振成像 脑梗死 治疗 随访 Susceptibility weighted imaging Magnetic resonance imaging Cerebral infarction Treatment Following up
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共引文献101

同被引文献23

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