摘要
本实验采用肠淋巴管引流术，在失血性休克大鼠模型的基础上，观察高渗盐水治疗失血性休克过程中肠淋巴流量及其蛋白含量的变化，旨在探讨高渗盐水对失血性休克大鼠治疗的淋巴机制。结果表明，休克大鼠输入高渗盐水或生理盐水后，两组大鼠的血压，肠淋巴流量及其蛋白输出量均比休克明显升高，高渗盐水治疗组显著高于生理盐水对照组（Ｐ〈０．０１），而且治疗组大鼠肠淋巴流量及共蛋白输出量远远高于休克前水平（Ｐ〈０．０１）。
In order to investigate the lymphatic mechanism of therapeutic effect of hypertonic sodium chloride ( HSC) on hemorrhagic shock. A cannula of intestinal lymph duct was used, intestinal lymph flow were measurable collected and recorded with a measurable cintainer and lymph concentration was determinded by Folin's method. The results showed that after the administration of HSC, the blood pressure, the lymph flow and lymph protein output in the treatment group were increased significantly compared with shock phase ( P<0.01) , and were higher obviously than that of control group( P< 0. 01); at the same time, the lymph flow and protein output in treatment group were enormously much more than that of pre - shock phase. These results suggested that the intestinal lymph circulation play an active role in the treatment of HSC on hemorrhagic shock.
