摘要
有关灼口综合征(BMS)的发病机理目前尚未确定。本文拟通过测定唾液谷胱甘肽过氧化物酶(GPx)的活性,来了解BMS患者口腔粘膜氧自由基(OFR)代谢状况的变化,从而探讨BMS的发病机理。结果显示:BMS组和正常组唾液GPx活性分别为9.30×0.39u/L和8.96±0.29u/L,两者差异无显著性(P>0.05)。
The pathogenesis of burning mouth syndrome (BMS) is still unclear. In this paper, glutathione peroxidase (GPx) level in saliva of patients was BMS was assayed in order to show the changes of oxygen free radicals (OFR) levels in BMS oral mucosa. So the pathogenesis of BMS might be explained. The result showed that the GPx activities in saliva of BMS group and normal group were 9. 30±0. 39u/L and 8. 96±0. 29u/L respectively. The difference between the two groups was not significant (P>0. 05).
出处
《临床口腔医学杂志》
1998年第1期35-36,共2页
Journal of Clinical Stomatology
