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晚期糖基化终产物及其受体在糖尿病周围神经病变中的作用 被引量：11

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摘要 该文概述了晚期糖基化终产物(AGEs)及其受体在周围神经组织中的表达与分布及其在糖尿病周围神经病变(DPN)中的作用。AGEs及其受体在DPN中的可能作用机制研究有望为开发预防、治疗DPN的新药提供理论基础。
作者 陆邦超 王坚
机构地区 南京军区南京总医院内分泌科
出处 《中国神经免疫学和神经病学杂志》 CAS 2007年第6期367-369,共3页 Chinese Journal of Neuroimmunology and Neurology
关键词 晚期糖基化终产物 糖尿病周围神经病变
分类号 R745 [医药卫生—神经病学与精神病学]
  • 相关文献

参考文献12

  • 1潘晓东,陈晓春.非酶糖化、晚期糖基化终产物与阿尔茨海默病[J].国际神经病学神经外科学杂志,2005,32(4):334-337. 被引量：3
  • 2Kikuchi S, Shinpo K, Takeuchi M, et al. Glycation-a sweet tempter for neuronal death[J]. Brain Res Reviews, 2003, 41:306-323.
  • 3Stracke H, Hammes HP, Werkmann D, et al. Efficacy of benfotiamine versus thiamine on function and glycation products of peripheral nerves in diabetic rats [J]. Exp Clin Endocrinol Diabetes, 2003,109:330-336.
  • 4Haslbeck KM, Schleicher ED, Friess U, et al. N(epsilon)-Carboxy-methyllysine in diabetic and non-diabetic polyneuropathies[J]. Acta Neuropathol, 2002, 104:45-52.
  • 5Wada R, Nishizawa Y, Yagihashi N, et al. Effects of OPB-9195, anti-glycation agent,on experimental diabetic neuropathy[J]. Eur J Clin Invest,2001,31:513-520.
  • 6Poirier O, Nicaud V, Vionnet N, et al. Polymorphism screening of four genes encoding advanced glycation end-product putative receptors.Association study with nephropathy in type 1 diabetic patients[J].Diabetes, 2001,50:1214-1218.
  • 7Takeuchi M. Neurotoxicity of advanced glycation end-products for cultured cortical neurons[J]. J Neuro-pathol Exp Neurol,2000,59:1094-1105.
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二级参考文献21

  • 1Picklo M J, Montine TJ, Amarnath V, et al. Carbonyl Toxicology and Alzheimer' s Disease. Toxicol Appl Pharmacol, 2002, 184(3): 187-197.
  • 2Kikuchi S, Shinpo K, Takeuchi M, et al. Glycation-a sweet tempter for neuronal death. Brain Res Reviews, 2003, 41 (2-3) : 306-323.
  • 3Takcuchi M, Yamagishi S, TAGE (toxic AGEs) Hypothesis in various chronic diseases. Med Hypotheses, 2004, 63 (3) : 449-452.
  • 4Steru D, Yan SD, Yah SF, et al. Receptor for advanced glycation endprodacts: a multiligand receptor magnifying cell stress in diverse pathologic settings. Adv Drug Deliv Rev, 2002, 54(12) : 1615-1625.
  • 5Chou DK. H, Zhang JH, Smith FI, et al. Developmental expression of receptor for advanced glycationend products (RAGE),amphoterin and sulfoglucuronyl ( HNK-1 ) carbohydrate in mouse cerebellum and their role in neuriteoutgrowth and cell migration. J Neurochem, 2004, 90(6): 1389-1401.
  • 6Mukherjee TK, Mukhopadhyay S, Hoidal JR. The role of reactive oxygen species in TNFα-dependent expression of the receptor for advanced glycation end products in human umbilical vein endothelial cells. Biochim Biophys Acta, 2005, 1744(2) : 213-223.
  • 7Sekido H, Suzuki T, Jomori T, et al. Reduced cell replication and induction of apoptosis by advanced glycation end products in rat Schwaun cells. Biochem Biophys Res Commun, 2004, 320 1 ) : 241-248.
  • 8Yeh CH, Sturgis L, Haidacher J, et al. Requirement for p38 and p44/p42 mitogen-activated protein kinases in RAGE mediated nuclear faetor-κB transcriptional activationand cytokine secretion. Diabetes, 2001, 50(6): 1495-1504.
  • 9Shuvaev VV, Laffont L.Serot Jean-Marie, et al. Increased protein glycation in cerebrospinal fluid of Alzheimer' s disease. Neurobiol Aging. 2001, 22(3): 397-402.
  • 10Mruthinti S, Buccafusco JJ, Hill WD, et al. Autoimmunity in Alzheimer's disease: increased levels of circnlating IgGs binding Aβand RAGE peptides. Neurobiol Aging, 2004, 25 (8): 1023-1032.

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同被引文献128

引证文献11

二级引证文献77

中国神经免疫学和神经病学杂志
2007年 第6期

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