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基质金属蛋白酶与心脏重构及心肌病理损害的研究进展 被引量:12

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出处 《中华老年心脑血管病杂志》 CAS 北大核心 2007年第7期499-500,共2页 Chinese Journal of Geriatric Heart,Brain and Vessel Diseases
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参考文献10

  • 1Lee RT,Lammerding J.Signaling pathways that influence extracellular remodeling[J].J Card Fail,2002,8:339-343.
  • 2柯丹,许春萱,林亚洲,张建成,陈林,林立芳,胡锡衷.心房颤动患者心房纤维化分子基础的临床研究[J].中华心血管病杂志,2005,33(5):459-463. 被引量:25
  • 3Fedak PW,Altamentova SM,Weisel RD,et al.Matrix remodeling in experimental and human heart failure:A possible regulatory role for TIMP-3[J].Am J Physiol Heart Circ Physiol,2003,284:626-634.
  • 4Khan A,Moe GW,Nili N,et al.The cardiac atria are chambers of active remodeling and dynamic collagen turnover during evolving heart failure[J].J Am Coll Cardiol,2004,43:68-76.
  • 5Kaden JJ,Dempfle CE,Sueselbeck T,et al.Time-dependent in the plasma concentration of matrix metalloproteinase 9 after acute myocardial infarction[J].Cardiology,2003,99:140-144.
  • 6郭建中,高长青.基质金属蛋白酶在大鼠心肌梗死模型心室重塑中的作用[J].临床心血管病杂志,2005,21(6):364-366. 被引量:7
  • 7Hayashidani S,Tsutsui H,Ikeuchi M,et al.Targeted deletion of MMP-2 attenuates early LV rupture and late remodeling after experimental myocardial infarction[J].Am J Physiol Heart Circ Physiol,2003,285:1229-1235.
  • 8Li J,Schwimmbeck PL,Tschope C,et al.Collagen degradation in a murine myocarditis model:relevance of matrix metalloproteinase in association with inflammatory induction[J].Cardiovac Res,2002,56:235-247.
  • 9朱慧,张薇,郭辰虹,张供,张维东,钟明,杨贵荣,葛志明,张运.心房肌基质金属蛋白酶-9和组织金属蛋白酶抑制因子-1表达改变对房颤心房重构机制的研究[J].中华医学杂志,2005,85(1):45-48. 被引量:31
  • 10Nakano Y,Niida S,Dote K,et al.Matrix metalloproteinase-9 contributes to human atrial remodeling during atrial fibrillation[J].J Am Coll Cardiol,2004,43:818-825.

二级参考文献30

  • 1Spinale FG, Coker ML, Bond BR, et al. Myocardial matrix degradation and metalloproteinase activation in the failing heart : a potential therapeutic target. Cardilvasc Res, 2000, 46:225-238.
  • 2Saharinen J, Hyytiainen M, Taipale J, et al. Latent transforming growth factor-beta binding proteins ( LTBPs ) structural extracellular nmtrix proteins for targeting TGF-beta action. Cytokine Growth Factor Rev, 1999, 10 : 99-117.
  • 3Peterson JT, Li H, Dillon L, et al. Evolution of matrix metalloproteinase and tissue inhibitor expression during heart failure progression int infracted rat. Cardiovasc Res, 2000, 46:307-315.
  • 4Nagahiro N, Kazuhiro Y, Yasushi S, et al. Differential activation of matrix metalloproteinases in heart failure with and without ventricular dilatation. Cardiovasc Res, 2003, 57:766-774.
  • 5Ducharme A , Frantz S, Aikawa M, et al. Targeted deletion of matrix metalloproteinase-9 attenuates left ventricular enlargement and collagen accumulation after experimental myocardial infarction. J Clin Invest, 2000, 106:55-62.
  • 6Heymans S, Luttun A, Nuyens D, et al. Inhibition of plasminogen activators or matrix metallproteinases prevents cardiac rupture but impairs therapeutic angiogenesis and causes cardiac failure . Nat Med, 1999, 5:1135-1142.
  • 7Borg TK, Burgess ML. Holding it all together: organization and function(s) of the extracellular matrix of the heart. Heart Failure,1993,8 : 230-238.
  • 8Li YY, Feldman AM, Sun Y, et al. Differential expression of tissue inhibitors of metalloproteinases in the failing human heart.Circulation, 1998, 98 : 1728-1734.
  • 9Roten L, Nemoto S, Simsic J, et al. Effects of gene diletion of the tissue inhibitor of the matrix metalloproteinase-type 1 ( TIMP-1 ) on left ventricular geometry and function in mice. J Mol Cell Cardiol,2000, 32:109-120.
  • 10Frustaci A, Chimenti C, Bellocci F, et al. Histological substrate of atrial biopsies in patients with lone atrial fibrillation . Circulation, 1997, 96: 1180-1184.

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引证文献12

二级引证文献72

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