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足细胞病的病因分类及诊治 被引量:3

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作者 周建华
出处 《实用儿科临床杂志》 CAS CSCD 北大核心 2006年第17期1123-1124,共2页 Journal of Applied Clinical Pediatrics
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参考文献12

  • 1Barisoni L,Mundel P.Podocyte biology and the emerging understanding of podocyte diseases[J].Am J Nephrol,2003,23(5):353-360.
  • 2Kriz W.Podocyte is the major culprit accounting for the progression of chronic renal disease[J].Microsc Res Tech,2002,57 (4):189-195.
  • 3Pavenstadt H,Kriz W,Kretzler M.Cell biology of the glomerular podocyte[J].Physiol Rev,2003,83 (1):253-307.
  • 4Pollak MR.The genetic basis of FSGS and steroid-resistant nephrosis[J].Semin Nephrol,2003,23(2):141-146.
  • 5Moudgil A,Nast CC,Bagga A,et al.Association of parvovirus B19 infection with idiopathic collapsing glomerulopathy[J].Kidney Int,2001,59(16):2126-2133.
  • 6Markowitz GS,Appel GB,Fine PL,et al.Collapsing focal segmental glomerulosclerosis following treatment with high-dose pamidronate[J].J Am Soc Nephrol,2001,12(6):1164-1172.
  • 7Petermann AT,Hiromura K,Blonski M,et al.Mechanical stress reduces podocyte proliferation in vitro[J].Kidney Int,2002,61 (1):40-50.
  • 8Kim YH,Goyal M,Wharram B,et al.GLEPP1 receptor tyrosine phosphatase(Ptpro) in rat PAN nephrosis:A marker of acute podocyte injury[J].Nephron,2002,90(4):471-476.
  • 9Nakamura T,Ushiyama C,Suzuki S,et al.The urinary podocyte as a marker for the differential diagnosis of idiopathic focal glomerulosclerosis and minimal-change nephrotic syndrome[J].Am J Nephrol,2000,20(3):175-179.
  • 10Durvasula RV,Shankland SJ.Podocyte injury and targeting therapy:An update[J].Curr Opin Nephrol Hypertens,2006,15 (1):1-7.

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