摘要
很多有机磷化合物除了易使人和动物产生急性中毒外，还能引发一种迟发性毒性，即“有机磷 化合物诱导的迟发性多发性神经病（ｏｒｇａｎｏｐｈｏｓｐｈａｔｅ－ｉｎｄｕｃｅｄ ｄｅｌａｙｅｄ ｐｏｌｙｎｅｕｒｏｐａｔｈｙ．ＯＰＩＤＰ）”。本文综述了有关ＯＰＩＤＰ的症状学、病理学和生物化学的研究及其进展。
Delayed polyneuropathy can be caused by many organophosphorus compounds in humans and some species of animals. This subject has been studied for more than 60 years. The symptomatology, pathology and biochemistry of organophosphate-induced delayed polyneuropathy (OPIDP) are reviewed in this paper.
出处
《动物学报》
SCIE
CAS
CSCD
1996年第4期436-443,共8页
ACTA ZOOLOGICA SINICA
关键词
迟发性神经病
有机磷化合物
迟发性神经毒性
Organophosphorus compounds (OPs), Delayed neurotoxicity, Neuropathy target enzyme (NTE), Calcium/calmodulin protein kinaseⅡ (CCPK Ⅱ).
