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黄体酮对创伤性脑损伤后炎症因子的抑制作用的研究 被引量:4

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摘要 创伤性脑损伤(Traumatic brain injury,TBI)后的炎症反应是导致继发性脑损害的主要原因之一。在TBI发生后早期,即有大量致炎细胞因子的释放和免疫细胞的激活。免疫细胞的激活,反过来又会导致自由基和更多的炎症介质释放。无论这种炎症级联反应由何种因素启动,对炎症反应的抑制都可以减轻脑水肿,减少神经元的死亡或胶质增生。
出处 《中华神经外科杂志》 CSCD 北大核心 2006年第3期186-189,共4页 Chinese Journal of Neurosurgery
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参考文献10

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同被引文献43

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  • 3潘德生,刘伟国,杨小锋.黄体酮对大鼠脑损伤后脂质过氧化的抑制作用[J].中华创伤杂志,2006,22(11):866-867. 被引量:4
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