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门静脉高压症形成的细胞分子机制 被引量：6

Cellular and molecular mechanism of portal hypertension
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摘要 门静脉高压症是一组由门静脉压力增高引起的症候群,绝大多数患者由肝硬化引起.近10年来门脉高压症的发病机制研究在病理生理学、病理学以及细胞分子理论方面取得了很大进展,其中门静脉高压症的细胞分子机制是当今研究的热点.
作者 王春平 韩军
机构地区 解放军第三○二医院
出处 《传染病信息》 2005年第3期117-119,共3页 Infectious Disease Information
关键词 细胞分子机制 门静脉高压症 发病机制研究 门静脉压力 病理生理学 门脉高压症 症候群 肝硬化 病理学
分类号 R512.62 [医药卫生—内科学] R657.34 [医药卫生—外科学]
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参考文献7

  • 1Gupta TK, Toruner M, Chung MK, et al. Endothelial dysfunction and decreased production of nitric oxide in the intrahepatic microcirculation of cirrhotic rats. Hepatology, 1998, 28(4):926-931
  • 2Reynaert H, Thompson MG, Thomas T, et al. Hepatic stellate cells:role in microcirculation and pathophysiology of portal hypertension.Gut, 2002, 50(4):571-581
  • 3Yang Y, Harvey SA, Gandhi CR. Kupffer cells are a major source of increased platelet activating factor in the CC14-induced cirrhotic rat liver, J Hepatol, 2003, 39(2):200-207
  • 4Yang Y, Harvey SA, Gandhi CR. Kupffer cells are a major source of increased platelet activating factor in the CC14-induced cirrhotic rat liver, J Hepatol, 2003, 39(2):200-207
  • 5Kureishi Y, Luo Z, Shiojima I, et al. The HMG-CoA reductase inhibitor simvastatin activates the protein kinase Akt and promotes angiogenesis in normocholesterolemic animals. Nat Med, 2000, 6(9):1004-1010
  • 6Yang Y, Nemoto EM, Harvey SA, et al. Increased hepatic platelet activating factor (PAF) and PAF receptors in carbon tetrachloride induced liver cirrhosis. Gut, 2004, 53(6):877-883
  • 7马雪梅,王春平,韩军,向轶,苏淑慧,冯永毅,杨永平.肝硬化形成过程中血小板活性因子及其受体在门脉高压形成中的意义[J].解放军医学杂志,2004,29(12):1062-1064. 被引量：6

二级参考文献9

  • 1姚希贤主编.临床消化病学.天津:天津科学技术出版社,1999.1056
  • 2Mark GC,Zhang JX.Regulation of sinusoidal perfusion: in vivo methodology and control by endothelins.Semin Liver Dis,1999,19(4):383
  • 3Alam I,Bass NM,Bacchetti P et al.Hepatic tissue endothelin-1 levels in chronic liver disease correlate with disease severity and ascites.Am J Gastroenterol,2000,95: 199
  • 4Yang YP,Harvey SA,Gandhi CR.Kupffer cells are a major source of increased platelet activating factor in the CCl4-induced cirrhotic rat liver.J Hepatol,2003,39(2): 200
  • 5Prescott SM,Zimmerman GY,Stafforini DM et al.Platelet-activating factor and relaed lipid mediators.Ann Rev Biochem,2000,69:419
  • 6Montrucchio G,Alloatti G,Camussi G.Role of platelet-activating factor in cardiovascular pathophysiology.Physiol Rev,2000,80:1669
  • 7Blank ML,Hall MN,Cress EA et al.Inactivation of 1-1lkyl-2-acetyl-sn-glycero-3-phosphocholine by a plasma acetylhydrolase: higher activities in hypertensive rats.Biochem Biophys Res Commun,1983,113:666
  • 8Chan CC,Hwang SJ,Lee FY et al.Prognostic value of plasma endotoxin levels in patients with cirrhosis.Scand J Gastroenterol,1997,32: 942
  • 9Meade CJ,Birke F,Metcalfe S et al.Serum PAF-acetylhydrolase in severe renal or hepatic disease in man: relationship to circulating levels of PAF and effects of nephrectomy or transplantation.J Lipid Mediat Cell Signal,1994,9(1): 205

共引文献5

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引证文献6

二级引证文献50

传染病信息
2005年 第3期

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