2Scorsin M, Hagege AA, Dolizy I, et al. Can cellular transplantation improve function in doxorubicininduced heart failure [ J ] .Circulation, 1998, 98( 19 Suppl): 151-155.
3Weissman IL. Stem cells-scientific, medical, and political issues[J]. N Engl J Med, 2002, 346(20): 1576-1579.
4Orlic D, Kaistura J, Ghimenti S, et al. Bone marrow cells regenerate infracted myocardium[J]. Nature, 2001, 410(6829):701-705.
5Mangi AA, Noiseux N, Kong D, et al. Mesenchymal stem cells modified with Akt prevent remodeling and restore performance of infarcted hearts[J]. Nat Med, 2003,9(9): 1195-1201.
6Makino S, Fukuda K, Miyoshi S, et al. Cardiomyocytes can be generated from marrow stromal cells in vitro[J]. J Clin Invest,1999,103 (5): 697-705.
7Asahara T, Masuda H, Takahashi T, et al. Bone marrow origin of endothelial progenitor cells responsible for postnatal vasculogenesis in physiological and pathological neovascularization[J ]. Circ Res,1999, 85(3) :221-228.
8Kawamoto A, Gwon HC, Iwaguro H, et al. Therapeutic potential of ex vivo expanded endothelial progenitor cells for myocardial ischemia[J]. Circulation, 2001,103(5) :634-637.
9Kocher AA, Schuster MD, Szabolcs MJ, et al. Neovascularization of ischemic myocardium by humanbone-marrow-derived angioblasts prevents cardiomyocyte apoptosis, reduces remodeling and improves cardiac function[J]. Nat Med, 2001, 7(9) :430-436.
10Kocher AA, Schuster MD, Szbolcs M J, et al. Neovascularization of ischemic myocardium by human bone-marrow-derived angioblasts prevents cardiomyocyte apoptosis, reduces remodeling and improves cardiac function[J]. Nat Med, 2001, 7(4) :430-436.
二级参考文献3
1Wakitani S,Saito T,Caplan AI.Myogenic cells derived from rat bone marrow mesenchymal stem cells exposed to 5-azacytidine[].Muscle and Nerve.1995
2Wang JS,Shumtim D,Galipeau J,et al.Marrow stromal cells for cellular cardiomyoplasty: feasibility and potential clinical advantages[].Journal of Thoracic and Cardiovascular Surgery.2000
3Tomita S,Li RK,Weisel RD,et al.Autologous transplantation of bone marrow cells improves damaged heart function[].Circulation.1999
