2[1]Rasmussen L, Oster JE, Qvist N, et al. The effects of omeprazole on intragastric pH, intestinal motility, and gastric emptying rate[J]. Scand J Gastroenterol,1999,34(7):671-675.
3[2]Fuchs KH, Fein M, Maroske T, et al. The role of 24-hr gastric pH monitoring in the interpretation of 24-hr gastric bile monitoring for duodenogastric reflux[J]. Hepatogastroenterology,1999,46(25):60-65.
4[3]Kromer W, Horbach S, Luhmann R. Relative efficacies of gastric proton pump inhibitors: their clinical and pharmacological basis[J]. Pharmacology,1999,59(2):55-77.
5[4]Hasselgren G, Keelan M, Kirdeikis P, et al. Optimization of acid suppression for patients with peptic ulcer bleeding: an intragastric pH-metry study with omeprazole[J]. Eur J Gastroenterol Hepatol,1998,10(7):601-606.
6[5]Savarino V, Mela GS, Zentilin P, et al. Comparison of 24-hr control of gastric acidity by three different dosages of pantoprazole in patients with duodenal ulcer[J]. Aliment Pharmacol Ther,1998,12(12):1241-1247.
7[6]Nishioka K, Nagao T, Urushidani T. Correlation between acid secretion and proton pump inhibitiors omeprazole and pantoprazole. Biochem Pharmacol,1999,58(8):1349-1359.
8Karmeli F, Eliakim R, Okon E, et ah Somatostatin effectively prevents ethanol and NSAID induced gastric mucosal damage in rats.Dig Dis Sci, 1994,39:617.
9Dy NM, Gostout CJ, Balm RK. Bleeding from the endoscopicallyidentified Dieulafoy' s lesion of the proximal small intestine and colon. Am J Gastroenterol, 1995, 90 : 108-111.
10Jones SC, Axon AT. Bleeding peptic ulcer endo6copic and pharmacological management. Postgrad Med J, 1991,67 : 606-608.