1Phillips AO,Steadman R. Diabetic nephropathy:the central role of renal proximal tubular cells in tubulointerstitial injury. Histol Histopathol, 2002,17(1):247-52.
2Nangatu M. Mechanisms of tubulointerstitial injury in the kidney: final common pathways to end stage renal failure. Intern Med, 2004,43 (1): 9- 17.
3Sinniah R,Khan TN.Renal tubular basement membrane changes in tubulointerstitial damage in patients with glomerular diseases. Ultrastruct Pathol,1999,23(6) ,359 - 68.
4Morrisey K. Renal proximal tubular cell fibronectin accumulation in response to glucose is polyol pathway dependent. Kidney Int, 1999,55(1):160 - 7.
5Morrisey K. Decreased degradation of collagen and fibronectin following exposure of proximal cells to glucose. Exp Nephol,1999,7 ( 5 - 6 ): 449 -62.
6Chen S,Jim B. Diabetic nephropathy and transforming growth factor- beta:transforming our view of glomerulaosclerosis and fibrosis build - up.Semin Nephrol,2003,23(6):532 - 43.
7Fraser D. Independent regulation of transforming growth factor- beta1 transcription and translation by glucose and plate let - derived growth factor Am J Pathol,2002,161(3):1039 - 49.
8Wolf G. Molecular mechanisms of diabetic renal hypertention. Kidney Int,1999,56(2)393 - 405.
9Han K. High glucose stimulates proliferation and collagen type 1 synthesis in renal cortical fibrosis:mediation by autocrine activation of TGF- beta.J Am Soc Nephrol,1999,10(9): 1891 - 9.
10Pillips A,Janssen U, Floege J.Progression of diabetic nephropathy: Insights from cell culture studies and animal models. Kidney Blood Press Res, 1999,22(1-2):81-97.