1Veerhuis R,Janssen I,De Groot CJ,et al.Cytokines associated with amyloid plaques in Alzheimer's disease brain stimulate human glial and neuronal cell cultures to secrete early complement proteins,but not C1-inhibitor[J].Exp Neurol,1999,160(1):289-299.
2Bard F,Cannon C,Barbour R,et al.Peripherally administered antibodies against amyloid beta-peptide enter the central nervous system and reduce pathology in a mouse model of Alzheimer disease[J].Nat Med,2000,6(8):916-919.
3Neumann H.Control of glial immune function by neurons[J].Glia,2001,36(2):191-199.
4Webster SD,Yang AJ,Margol L,et al.Complement component C1q modulates the phagocytosis of Abeta by microglia[J].Exp Neurol,2000,161(1):127-138.
5Bergamaschini L,Donarini C,Rossi E,et al.Heparin attenuates cytotoxic and inflammatory activity of Alzheimer amyloid-beta in vitro[J].Neurobiol Aging,2002,23(4):531-536.
6Pike CJ.Estrogen modulates neuronal Bcl-xL expression and beta-amyloid-induced apoptosis:relevance to Alzheimer's disease[J].J Neurochem,1999,72(4):1552-1563.
7Campbell A.beta-amyloid: friend or foe[J].Med Hypotheses,2001,56(3):388-391.
8Popovic M,Caballero-Bleda M,Puelles L,et al.Importance of immunological and inflammatory processes in the pathogenesis and therapy of Alzheimer's disease[J].Int J Neurosci,1998,95(3-4):203-236.
9Bergamaschini L,Canziani S,Bottasso B,et al.Alzheimer's beta-amyloid peptides can activate the early components of complement classical pathway in a C1q-independent manner[J].Clin Exp Immunol,1999,115(3):526-533.
