1[1]Tobias PS,Soldau K,Ulevitch RJ.Isolation of a lipopolysaccharide binding acute phase reactant from rabbit serum[J]. J Exp Med,1986,164:777-793.
2[2]Heumann D,Roger T. Initial responses to endotoxins and gramnegative bacteria[J]. Clin Chimica Acta,2002,323:59-72.
3[3]Triantafilou M,Triantafilou K. Lipopolysaccharide recognition:CD14,TLRs and the LPS-activation cluster[J]. Trends Immunol,2002,23:301-304.
4[4]Cheung DO,Halsey YK, Spreert DP. Role of pulmonary alveoliar macrophages in defense of the lung against pseudomonas aeruginosa[J]. Infect Immun,2002,4585-4592.
5[5]Zhang P,Bagby GJ,Kolls, et al. The effects of granulocyte colony-stimulating factor and nertrophil recruitment on the pulmonary chem.okine response to intratracheal endotoxin[J]. J Immunol, 2001,165:458-465.
6[6]Sunil VR,Connor AJ,Guo Y,et al.Activation of type ialveolar epithelial cells during acut endotoxemia[J].Am J Physiol Lung Cell Mol Physiol,2002,282:L872-L880.
7[7]Dentener MD,Vreugdenhil ACE,Hoet PHM,et al. Production of the acute phase protein LPS binding protein (LBP) by respiratory type II epithelial cells:implications for local defense to bacterial endotoxins[J].Am J Res Cell Mol Biol,2000,23:146-153.
8[8]Browder W,Ha T,Chuanfu L,et al.Early activation of pulmonary nuclear factor κB and nuclear factor interleukin-6 in polymicrobial sepsis[J].J Trauma,1999,46(4):590-596.
9[9]Lee JI, Burckart GJ.Nuclear factor kappaB:Important transcription factor and therapeutic target[J].J Clin Pharmacol,1998,38(11):981-993.
10[10]Patrick A,Baltimore B,Baltimore D.NF-κB:ten years after[J]. Cell,1996,87(1):13.