摘要
抑菌测定结果表明:在改良的Kelman平板培养基上,1～20mmol的阿魏酸、香豆酸、香草酸、肉桂酸、磺基水杨酸、对羟基苯甲酸和水杨酸钠对青枯病菌的生长没有影响,因此在用水杨酸钠淋根后,植株抗病性的增强是由于桉树苗木产生诱导抗病性的结果,而不是该化合物的直接毒性;浓度为5～10mmol的水杨酸钠均可以获得较强的诱导效果,并以接种前5～7d处理桉树苗的诱导抗病性最强;其它的芳香酸对桉树抗青枯病没有诱导活性.
The results from in vitro toxicity test indicate that 1 ~ 20 mmol of ferulic acid, coumaric acid, vanillic acid, cinnamic acid, sulfonic salicylate, phydroxybenzoic acid and sodium salicylate have no inhibition over the growth of Ralstonia solanacearum. Disease reduction of eucalypt bacterial wilt was only observed by pouring 10 mL of sodium salicylate to the root of Eucalyptus urophylla. The concentration of sodium salicylate from 1 mmol to 10 mmol can induce systemically acquired resistance against Ralstonia solanacearum in eucalypt, but the best disease resistance induction can be obtained with 5 ~ 10 mmol of sodium salicylate 5 to 7 days prior to challenging inoculation with pathogen. The other phenolic acids tested have no inducing effect on disease resistance at all.
出处
《中南林学院学报》
CSCD
2003年第4期35-38,57,共5页
Journal of Central South Forestry University
基金
国家自然科学基金(编号:39970615)
中南林学院青年科学基金资助.
关键词
桉树
青枯病
芳香酸
诱导抗病性
阿魏酸
香豆酸
香草酸
肉桂酸
磺基水杨酸
forest protection
phenolic acids
sodium salicylate
Eucalyptus urophylla
bacterial wilt
systemically acquired resistance
