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病毒性脑炎患儿血清和脑脊液sICAM-1和TNF-α的变化及意义 被引量:1

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出处 《山东医药》 CAS 北大核心 2003年第13期39-40,共2页 Shandong Medical Journal
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  • 2GlimakerM 赵杨.各种不同病因的脑膜炎患者脑昔藏中的肿瘤坏死因子-α(TNF-α)[J].国外医学:临床生物化学与检验学分册,1994,15:190-191.
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  • 1董胜英,布月青,孙红娟.病毒性脑炎患儿血清、脑脊液TNF-α和sICAM-1的变化[J].实用医学杂志,2005,21(11):1180-1181. 被引量:3
  • 2Pietruczuk M, Pietruczuk A, Pancewicz S, et al. ICAM1: structure, biological role and clinical significance. Pol Merkuriusz Lek, 2004, 17(101):507-511.
  • 3Marker O,Seheynius A,Christensen JP, et al. Virus-activated T cells regulate expression of adhesion molecules on endothelial cells in sites of infection. J Neuroimmunol,1995,62(1):35-42.
  • 4Christensen JP, Johansen J, Marker O, et al. Circulating intercellular adhension molecules-1 (ICAM-1) as an early and sensitive marker for virus-induced T cell activation. Clin Exp Immunol, 1995,102(2):268-273.
  • 5Sanchez-Burgos G, Hernandez-Pando R, Campbell IL, et al. Cytokine production in brain of mice experimentally infected with dengue virus. Neuroreport, 2004, 15(1): 37-42.
  • 6Lopez S, Prats N, Marco A. Expression of E-selection, P-seelection, P-selection , and intercellular adhesion molecule-1 during experimental murine Itsteriosis. Am J Pathol,1999,155(4):1391-1397.
  • 7Freyer D, Manz R, Ziegelom A, et al. Cerebra endothelial cells release TNF-α after stimulation with cell walls of Streptococcus pneumoiae and regulate inducible nitric oxide synthase and ICAM-1 expression via autocrine loops. J Immunol, 1999,163(8): 4308-4314.
  • 8芦军萍,王怀立.黏附分子与中枢神经系统感染[J].国外医学(儿科学分册),2002,29(2):70-73. 被引量:8

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