1Holmin S, Malldesen T. Intracerebral sdministration of interleukin- 1β and induction of inflammation, apoptosis, and vasogenic edema. J Neurosura.2000.92(1) : 108-120.
2Stover JF, Schoning B, Beyer TF, et al. Temporal profile of cerebrospinal fluid glutamate, intcrleukin - 6 and tumor necrosis factor -αin relation to brain edema and contusion following controlled cortical ingmct injury in rats. NeurosciLett,2000,288(1) :25 - 28.
5Cemak I, Chapman S, Hamlin G,et al.Temporal characterisation of proand an ti - apoptotic mechanisms following diffuse traumatic brain injury in rats.J Clin Neurasci,2002,9(5) :565.
6Asai A, Qiu JH, Narita Y. et al. High level calcineurin activity predisposes neuronal cells to apoptosis. J Biol Chem, 1999, Nov 26;274(48):34450- 34458.
7Aronowski J, Grotta JC, Strong R. et al. Inteaplay between the gamma isoform ff PKC and calclneurin in regulation of vulnerability to focal cerebral ischemia. J Cereb Blood Flow Metab,2000,20(2):343- 349.
8Alexanian AR, Bamburg JR. Neuronal survival activity of s100ββ enhanced by calcineurin inhibitors and requires activation d NF - кB.FASEB J, 1999,13(12) : 1611 - 1620.
9Genazzani AA, Carafoli E,Guerini D. Cslcineurin controls ionsitol 1,4,5- trisphosphate type Ⅰ reeoptor expression in neurons. Proe Natl Aead Sei USA, 1999,96(10) :5797 - 5801.
10Morioka M, Hamada JI, Ushio Y, et al. Potential role of calcineurin for brain ischemia and traumatic injury. Prog Neurobiol, 1999, 58 ( 1 ) : 1 -30.
