缬沙坦和依那普利逆转高血压患者左室肥厚的临床观察

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摘要血管紧张素转换酶抑制剂(ACEI)的降压及对靶器官的保护作用已得到公认[1],其作用是通过抑制转换酶的活性,减少血管紧张素(Ang)Ⅰ转换为AngⅡ,从而阻断AngⅡ的作用而发挥疗效.

作者朱振霞林吉秋

机构地区烟台山医院心血管科烟台市电力局卫生所

出处《中国煤炭工业医学杂志》 2003年第4期366-366,共1页 Chinese Journal of Coal Industry Medicine

关键词缬沙坦依那普利高血压病左心室肥厚

参考文献5

1[1]Ennis I L, Alvarez B V, Camilion D H M C, et al. Enalapril induces regression of cardiac hypertrophy and normalization of PH:regulatory mechanisms. Hypertention, 1998, 31(4) :961-967
2[2]Timmermans PBMWM, Wong PC, Chiu AT, et al. The preclinical basis of the therapeutic evaluation of losartan. J Hypertens,1995,13(Suppl. 1): 1-13
3[3]Ling Q, Guo Z G, Su Z, et al. Regression of cardiac hypertrophy and myosin isoenzyme patterns by losartan and Captopril in renovascular hypertensive rats. Acta Pharmacol Sin, 1994, 15: 206-210
4[4]Sadoshima J, IzumoS S. Signal transsuction pathways of angiotension Ⅱ induced c- fos gene expression in cardiac myocytes in vitro. circ Res, 1993, 73(3) :424-430
5[5]Schieffer B, Wirger A, Meybrunn M, et al. Comparative effects of chronic angiotens in - converting enzyme inhibition and angiotensin Ⅱ type 1 receptor blockade on cardiac remodeling after myocardial infarction in the rat. Circulation, 1994, 89: 2273-2282

中国煤炭工业医学杂志

2003年第4期

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