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基于虚、毒、瘀理论探讨痛风的发病机制 被引量:2

Exploring Pathogenesis of Gout Based on Theory of Deficiency,Toxin and Stasis in Traditional Chinese Medicine
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摘要 痛风在过去称为“富贵病”“贵族病”,目前是一种常见的风湿病。随着社会生活水平的提升,痛风发病越来越年轻化。中医历代医家对痛风认识深刻,中医治疗痛风疗效显著。根据中医文献和多年临证经验,总结出脾肾亏虚是痛风发病的根本原因,毒热是痛风发病的重要条件,毒蕴血瘀是痛风发病的病机关键,内外相引为发为痛风。文章通过详细阐述虚、毒、瘀理论应用于痛风病机,以期更好地指导临床中医、中西结合治疗痛风。 Gout used to be called“rich disease”and“noble disease”.Nowadays,gout is a common rheumatic disease.With the improvement of social living standards,the incidence of gout is becoming younger and younger.Doctors of traditional Chinese medicine have a profound understanding of gout,and the curative effect of traditional Chinese medicine in the treatment of gout is remarkable.According to the records of gout in traditional Chinese medicine literature and years of clinical experience of the disease,it is concluded that spleen and kidney deficiency is the root cause,heat toxin is an important condition for the onset,the accumulation of toxin and blood stasis is the key to the pathogenesis,both the internal and external causes lead to gout.In the article,the theory of deficiency,toxin and stasis in traditional Chinese medicine is applied to the mechanism of pathogenesis of gout in detail,in order to better guide the clinical treatment of gout both in traditional Chinese medicine and the combination of traditional Chinese and Western medicine.
作者 任妮娜 凌益 马武开 曾苹 姚血明 REN Nina;LING Yi;MA Wukai;ZENG Ping;YAO Xueming(Guizhou University of Traditional Chinese Medicine,Guiyang 550002,Guizhou,China;The Second Affiliated Hospital of Guizhou University of Traditional Chinese Medicine,Guiyang 550001,Guizhou,China)
出处 《辽宁中医杂志》 北大核心 2025年第8期44-47,共4页 Liaoning Journal of Traditional Chinese Medicine
基金 国家自然科学基金项目(81660811) 贵州省高层次创新型人才培养计划—“百”层次人才项目(黔科合平台人才[2016]5650) 贵州省中医风湿免疫病临床研究中心项目(黔科合平台人才[2020]2202号) 贵州中医药大学国家与省级科技创新人才团队培育项目(贵中医TD合字[2022]004号)。
关键词 痛风 毒蕴血瘀 因虚致实 脾肾亏虚 中医药 发病机制 gout accumulation of toxin and blood stasis excess syndrome caused by deficiency spleen and kidney deficiency traditional Chinese medicine pathogenesis
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