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“毒瘀损络”理论在消渴目病中的运用 被引量:7

Application of"Toxin and Stasis Injuring Collateral"Theory in Eye Disease of Diabetes Patients
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摘要 消渴日久,脏腑耗损,气阴两虚,精微化生过剩,津液输布失常,内生糖毒、浊毒、热毒,"毒"淫机体,挟糖毒耗损气血,挟浊毒黏滞缠绵,挟热毒灼营伤阴,致使血行不畅,"瘀"从"毒"化,共损目络,而成消渴目病。"毒瘀损络"为消渴目病的关键病机,解毒化瘀是治疗本病的基本方法。解毒通络使水谷精微得以运化,津液输布恢复正常,五脏六腑、四肢百骸受其濡养,从而改善消渴目病诸症。针对毒之成因和毒之特性,在解毒通络的同时,结合益气、养阴、化痰、清热等法治病求本,扶正祛邪。健脾益气使脾气强健,气血化生源源不息;养阴生津使阴精充足,肝肾之精源源不绝;祛除痰浊、清热解毒,使营阴充足不黏稠、血行流利不成瘀。活血化瘀法应贯穿本病的治疗始终。 When diabetes lasts for a long time,patients′Zang and Fu is worn out with Qi and Yin deficiency.With too much metaplasia of the subtle nutrition and abnormal fluid delivery,endogenous sugar toxin,turbidity,and Heat toxin are produced,which would injure Qi and Blood,and with time result in poor blood flow,namely,Blood stasis.This would damage the eye collateral and cause eye disease,being the key pathogenesis of eye disease of diabetes patients.Therefore,detoxification and dispersing stasis is the basic method of the treatment.Detoxification and dredging collaterals enable the subtle nutrition reach the Zang and Fu and support all parts of the body so as to improve the symptoms of the eye diseases.Besides,it should be combined with tonifying Qi,nourishing Yin,reducing Phlegm and clearing Heat.Invigorating the Spleen and strengthening Qi offers sources of vitality,while nourishing Yin would increase the essence of the Liver and Kidney.The method of promoting blood circulation and removing blood stasis would better the blood condition,so it should be used throughout the treatment of this disease.
作者 李方怡 徐寒松 陈永华 倪洪岗 李丹 LI Fangyi;XU Hansong;CHEN Yonghua;NI Honggang;LI Dan Guizhou(University of Traditional Chinese Medicine,Guiyang Guizhou China 550025)
机构地区 贵州中医药大学
出处 《中医学报》 CAS 2020年第8期1627-1630,共4页 Acta Chinese Medicine
基金 国家自然科学基金项目(81260600,81560733,81760813) 贵州省优秀青年科技人才专项项目{黔科合人字[2013]49号} 贵州省科技合作计划项目{黔科合LH字[2016]7127号} 贵州省苗医药重点实验室开放项目{黔苗医药K字[2017]013}。
关键词 消渴目病 “毒瘀损络” 糖毒 浊毒 热毒 解毒化瘀 eye disease of diabetes patients "toxin and stasis injuring collateral" sugar toxin turbid toxin Heat toxin detoxification and dispersing stasis
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