1Rohde LE,Ducharme A,Arroyo LH,et al.Matrix metalloproteinase inhibition attenuates early left ventricular enlargement after experimental myocardial infarction inmice[].Circulation.1999
2Hojo Y,Ikeda U,Ueno S,et al.Expression of matrix metalloproteinases in patients with acute myocardial infarction[].Japanese Circulation Journal.2001
3Liu YE,Wang M,Greene J,et al.Preparation and characterization of recombinant tissue inhibitor of metalloproteinase-4 (TIMP-4 )[].Journal of Biological Chemistry.1997
4Nagase H.Activation mechanisms of matrix metalloproteinase[].Biological Chemistry.1997
5Ye S.Polymorphism in matrix metalloproteinase genepromotors:implication in regulation of gene expression and susceptibility of various diseases[].Matrix Biology.2000
6Li YY,Feldman AM,Sun Y,et al.Differential expression of tissue inhibitors of metalloproteinases in the failing human heart[].Circulation.1998
7Sato H,Kinoshita T,Takino T,et al.Activation of a recombinant membrane type 1-matrix metalloproteinase(MT1-MMP) by furin and its interaction with tissue inhibitor of metalloproteinases (TIMP)-2[].FEBS Letters.1996
8Rouet-Benzineb P,Gontero B,Dreyfus P,et al.AngiotensinⅡ induces nuclear factor-κB activation in cultured neonatal rat cardiomyocytes through protein kinase C signaling pathway[].Journal of Molecular and Cellular Cardiology.2000
9Wassenaar A,Verschoor T,Kievits F,et al.CD40 engagement modulates the production of matrix metalloproteinases by gingival fibroblasts[].Clinical and Experimental Immunology.1999
10Spinale FG,Coker ML,Heung LJ,et al.A matrix metalloproteinase Induction/activation system exists in the human left ventricular myocardium and is upregulated in heartfailure[].Circulation.2000
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1Rutschow S,Leschka S,Westermann D,et al.Left ventricular enlargement in coxsackievirus-B3 induced chronic myocarditisongoing inflammation and an imbalance of the matrix degrading system.Eur J Pharmacol,2010,630:145-151.
2Miyazaki S,Kasai T,Miyauchi K,et al.Changes of matrix metalloproteinase-9 level is associated with left ventricular remodeling following acute myocardial infarction among patients treated with trandolapril,valsartan or both.Circ J,2010,74:1158-1164.
3Truter SL,Catanzaro DF,Supino PG,et al.Differential expression of matrix metalloproteinases and tissue inhibitors and extracellular matrix remodeling in aortic regurgitant hearts.Cardiology,2009,113:161-168.
4Matsumoto Y,Tsukada Y,Miyakoshi A,et al.C protein-induced myocarditis and subsequent dilated cardiomyopathy:rescue from death and prevention of dilated cardiomyopathy by chemokine receptor DNA therapy.J Immunol,2004,173:3535-3541.
5Cha MC,Purslow PP.Matrix metalloproteinases are less essential for the in-situ gelatinolytic activity in heart muscle than in skeletal muscle.Comp Biochem Physiol A Mol Integr Physiol,2010,156:518-522.
6Shimada K,Okabe TA,Mikami Y,et al.Therapy with granulocyte colony-stimulating factor in the chronic stage,but not in the acute stage,improves experimental autoimmune myocarditis in rats via nitric oxide.J Mol Cell Cardiol,2010,49:469-481.
7Matsumoto Y,Park IK,Kohyama K.B-cell epitode spreading is a critical step for the switch from C-protein-induced myocarditis to dilated cardiomyopathy.Am J Pathol,2007,170:43-51.
8Cai YH,Alvarez A,Alcaide P,et al.Abrogation of functional seletin-ligand expression reduces migration of pathogenic CD8 + T cell into heart.J Immunol,2006,176:6568-6575.
9Cheung C,Marchant D,Walker EK,et al.Ablation of matrix metalloproteinase-9 increases severity of viral myocarditis in mice.Circulation,2008,117:1574-1582.
10Golub LM,Ramamurthy NS,Llavaneras A,et al.A chemically modified nonantimicrobial tetracycline (CMT-8) inhibits gingival matrix metalloproteinase,periodontal breakdown,and extra-oral bone loss in ovariectomized rats.Ann N Y Acad Sci,1999,30:290-310.
