期刊文献+

早期肠内营养联合早期运动促进外科重症患者康复 被引量:5

暂未订购
导出
摘要 代谢是机体的基本功能之一,营养素是机体代谢功能的基本物质。重症患者由于营养摄入不足、丢失增加,加之创伤、感染导致的细胞因子和儿茶酚胺等分解代谢激素分泌增加,造成蛋白质等营养素消耗增多,营养不良发生率高达40%~100%。2015年欧洲肠外肠内营养学会(ESPEN)发表的专家共识提出了营养紊乱的概念及其诊断体系,
出处 《临床外科杂志》 2016年第12期960-962,共3页 Journal of Clinical Surgery
  • 相关文献

参考文献4

二级参考文献52

  • 1蹇英,朱雪芬,黄佳慧.综合呼吸功能锻练在肺移植术前病人中的应用[J].中国医药导报,2006,3(11). 被引量:9
  • 2黎介寿.肠功能障碍[J].中国胃肠外科杂志,1998,1(1):1-3. 被引量:9
  • 3Bodine SC, Stitt TN, Gonzalez M, et al. Akt/mTOR pathway is a crucial regulator of skeletal muscle hypertrophy and can prevent muscle atrophy in vivo. Nat Cell Biol. 2001 ; 3(11 ): 1014-1019.
  • 4Pallafacchina G, Calabria E, Serrano AL, et al. A protein kinase B-dependent and rapamycin-sensitive pathway controls skeletal muscle growth but not fiber type specification. Proc Natl Acad Sci U S A. 2002; 99(14):9213-9218.
  • 5Nader GA, McLoughlin T J, Esser KA.mTOR function in skeletal muscle hypertrophy: increased ribosomal RNA via cell cycle regulators.Am J Physiol Cell Physiol. 2005; 289(6):C1457-1465.
  • 6Stokoe D, Stephens LR, Copeland T, et al. Dual role of phosphatidylinositol-3,4,5-trisphosphate in the activation of protein kinase B. Science. 1997; 277(5325):567-570.
  • 7Schmelzle T, Hall MN. TOR, a central controller of cell growth. Cell. 2000; 103(2):253-262.
  • 8Ohanna M, Sobering AK, Lapointe T, et al.Atrophy of S6K1(-/-) skeletal muscle cells reveals distinct mTOR effectors for cell cycle and size control. Nat Cell Biol. 2005; 7(3):286-294.
  • 9Bodine SC.mTOR signaling and the molecular adaptation to resistance exercise.Med Sci Sports Exerc. 2006; 38(11):1950-1957.
  • 10Nave BT, Ouwens M, Withers D J, et al.Mammalian target of rapamycin is a direct target for protein kinase B: identification of a convergence point for opposing effects of insulin and amino-acid deficiency on protein translation.Biochem J. 1999; 344 Pt 2:427-431.

共引文献206

同被引文献40

引证文献5

二级引证文献8

相关作者

内容加载中请稍等...

相关机构

内容加载中请稍等...

相关主题

内容加载中请稍等...

浏览历史

内容加载中请稍等...
;
使用帮助 返回顶部