摘要
目的 总结急性完全性颈髓损伤继发低钠血症的变化规律并探讨其发生机制。方法 回顾分析 35例急性完全性颈髓损伤患者的血尿生化随时间变化规律。结果 本组低钠血症发生率 10 0 % ,低钠血症于伤后 4 .4 9d± 1.17d开始 ,13.6 1d± 3.0 7d达高峰 ,38.89d± 9.6 4 d恢复正常 ,持续时间 34.4 d± 9.5 d,15例 (42 .88% )出院时低钠血症仍未恢复。伤后平均 15 d左右血浆渗透压开始低于正常 ,此后一直持续至出院。结论 严重、顽固的低钠血症是颈髓损伤后极为常见的并发症 ,其发生机制可能与脑耗盐综合征 (CSWS)有关。
Objective To define the occurrence rate,time course,and potential mechanism of hyponatremia in patients after Frankel A Class acute cervical spinal cord injury.Methods Blood and urine of 35 hospitalized cases were analyzed retrospectively.Results Hyponatremia,whose occurrence rate was 100%,developed at a mean time of 4.49±1.17 days postinjury,reached its peak at 13.61±3.07 days and recovered to normal at 38.89±9.64 days. Fifteen (42.88%) cases did not recovered from hyponatremia during hospitalization.The plasma hypotonicity began to develop at a mean time of about 15 days after injury and then continued to discharge.Conclusion Severe and obstinate hypernatrmia were very common complications of cervical spinal cord injury,and the mechanism might be related to the cerebral salt wasting syndrome.
出处
《中国误诊学杂志》
CAS
2002年第7期968-970,共3页
Chinese Journal of Misdiagnostics
