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血清可溶性E-选择素与2型糖尿病关系探讨

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出处 《华夏医学》 2002年第1期66-67,共2页 Acta Medicinae Sinica
基金 广西区科技厅 2 0 0 0年广西科学研究与技术开发计划项目 (桂科攻 0 0 150 4 7)
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参考文献7

  • 1温玉洁.细胞粘附分子与糖尿病肾病的关系以及应用低分子肝素的治疗[J].广西医学,2000,22(3):541-545. 被引量:5
  • 2姜文宇.粘附分子与糖尿病慢性并发症[J].国外医学(内科学分册),1999,26(7):294-296. 被引量:31
  • 3Anitaben Tailor PhD,D. Neil Granger PhD. Role of adhesion molecules in vascular regulation and damage[J] 2000,Current Hypertension Reports(1):78~83
  • 4. Fosinopril decreases levels of soluble vascular cell adhesion molecule-1 in Type II diabetic patients suffering from microalbuminuria[J] 1998,Diabetologia(8):980~981
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  • 6L. Cominacini,A. Fratta Pasini,U. Garbin,M. Campagnola,A. Davoli,A. Rigoni,M. G. Zenti,A. M. Pastorino,V. Lo Cascio. E-Selectin plasma concentration is influenced by glycaemic control in NIDDM patients: possible role of oxidative stress[J] 1997,Diabetologia(5):584~589
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二级参考文献23

  • 1叶山东.糖尿病肾病的早期诊断[J].中华内分泌代谢杂志,1995,11(2):107-109. 被引量:49
  • 2邓义斌.整合素与糖尿病性肾病[J].国外医学(内分泌学分册),1996,16(4):181-184. 被引量:1
  • 3王竹兰 冯根宝 等.642例糖尿病患者肾脏病变的调查及其临床分析[J].中国糖尿病杂志,1995,3(1):7-7.
  • 4袁明霞.-[J].国外医学:眼科学分册,1998,22:50-54.
  • 5Steiner M, Reinhardt KM. Blann AD. Soluble adhesion molecules in NIDDM: increasedconcentration, relation to glycometablic control and possible pathophysiologicalsignificance [letter; comment]. Diabetologia 1996;39(7):868
  • 6Cominacini L, Fratta PA, Garbin U. et al.E-selectin plasma concentration isinfluenced by glycaemic control in NIDDM patient:possible role of oxidative stress.Diabetologia 1997;40(5):584
  • 7Albertini JP, Valensi P, Lormeau B, et al. Elevated concentration of solubleE-selection and vascular cell adhesion molecule-1 in NIDDM effect of intenisive insulintreatment. Diabetes Care 1998;21(6):1008
  • 8Thornhill MH, Wollicome SM, Mahiouz DL, et al. TNFcombines with IL 4 orinterferon-r to selectlively enhance endothelial cell adhesiveness to T-cell: thecontribution of VCAM-1 dependent and independent binding mechanism. J Immunol 1991;146:592
  • 9Martin S, Heidenthal E, schulte B, et al. Soluble forms of intercellular adhesionmolecule-1 inhibit insulitis and onset of autoimmune diabetes. Diabetologia1998;41(11):1298
  • 10Yagi N, Yokone K, Amano K, et al. Expression of Intercellular adhesion molecule 1on pancreatic β-cells accelerates β-celldestruction by cytotoxin T-cells in MurimeAutoimmune. Diabetes 1995;44:744

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