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柳氮磺胺吡啶对类风湿关节炎患者肿瘤坏死因子α水平的影响

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出处 《临床荟萃》 CAS 北大核心 2001年第14期646-647,共2页 Clinical Focus
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参考文献6

  • 1[1]Koopman WJ,Gay S.Do nonimmunologically mediated pathways play a role in the pathogenesis of rheumatoid arthritis? Rheum Dis Clin N Am,1993,(2):107-122
  • 2[2]Firestein GS.Invasive fibroblast-like synoviocytes in rheumatoid arthritis passive responders or transformed aggressor? Arthritis Rheum,1996,39(2):1781-1790
  • 3[3]Isomaki P,Luukkainen R,Toivanen P,et al.The presence of interleukin-13 in rheumatoid synovium and its antiinflammatory effects on synovial fluid macrophages from patients with rheumatoid arthritis.Arthritis Rheum,1996,39(2):1693-1702
  • 4[4]Grom AA,Murray KJ,Luyrink L,et al.Patterns of expression of tumor necrosis factor α,tumor mecrosis factor β,and their receptors in synovia of patients with juvenile spondylarthropathy.Arthrtis Rheum,1993,36(2):490
  • 5[5]Manicourt DH,Triki R,Fukuda K,et al.Levels of circulating tumor necrosis factor alpha and interleukin-6 in patients with rheumatoid arthritis.Arthritis Rheum,1993,36(2):490
  • 6[6]Barrera P,Boerbooms AMT,Janssen E,et al.Circulating soluble tumor necrosis factor receptors,interleukin-2 receptors,tumor necrosis factor alpha,and interleukin-6 levels in rheumatoid arthritis.Arthritis Rheum,1993,36(4):1070

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