1[1]Blackburn EH. Structure and function of telomeres. Nature, 1991,350: 569-573
2[2]Levy MZ,Zllsopp RC,Furcher B,et al. Telomere end-replication problem and cell aging. J Mol Biol, 1992,225: 951-960
3[3]Allsopp R, Chang E, Kashefi-Aazam M, et al. Telomere shortening is associated with cell division in vitro and in vivo. Exp Cell Res, 1995,220:194-200
4[4]Blackburn EH. Structure and function of telomeres. Nature, 1991,350: 569-573
5[5]Feng J, Funk WD, Wang S, et al. The RNA component of human telomer-ase. Science, 1995,269:1236-1241
6[6]Counter CM, Botelho FM, Wang P, et al. Stabilization of short telomeresand telomerase activity accompany immortalization of Epsteinbarr virus-transformed human B lymphocytes. J Virol, 1994,68: 3410-3414
7[7]Kim NW, Piatyszek MA, Prowse KR, et al. Specific association of humantelomerase activity with immortal cells and canaer. Science, 1994, 266:2001-2015
8[8]Counter CM, Hiret HW, Bocchett S, et al. Telomerase activity in humanovarian carcinoma. Proc Natl Acad Sci USA, 1994,91: 2900-2904
9[9]Hiraga S, Taranori O, Shuichi I, et al. Telomerase activity and alteratiousin telomere length in human brain tumors. Cancer Res, 1998,58: 2117-2125
10[10]Landberg G, Nielsen NH, Nilsson P, et al. Telomcrase ativity is associatedwith cell cycle deregulation in human breast cancer. Cancer Res, 1997,57: 549-554