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碘与甲状腺疾病 被引量:2

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出处 《医师进修杂志》 北大核心 2001年第7期3-5,共3页 Journal of Postgraduates of Medicine
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  • 1罗玉玉,叶振坤,项建梅,阎玉芹,陈祖培.碘酸钾和碘化钾治疗大鼠缺碘性甲状腺肿的体视学研究[J].中国体视学与图像分析,2003,8(1):16-19. 被引量:4
  • 2王连方.碘的生物学基础及其与健康关系[J].地方病通报,1994,9(3):72-76. 被引量:6
  • 3Burikhanov RB, Matsuzaki S. Excess iodine induces apoptosis in the thyroid of goitrogen-pretreated rots in vivo[J]. Thyroid, 2000,10:123-129.
  • 4Feldkamp J,Pascher E,Pemiok A,et al. Fas mediated apoptosis is inhibited by TSH and iodine in moderate concentration in primary human thyrocytes in vitro[ J ]. Hrom Metab Res, 1999,31:355-358.
  • 5Tamura M, Kimura H, Koji T, et al. Role of apoptosis of thyrocytes in a rat model of goiter: a possible involvement of Fas systerm[J]. Endocrinology,1998,193 : 3646-3653.
  • 6Bagchi N, Brown TR, Sundick RS. Thyriod cell injury is an initial events in the induction of autoimmune thyroiditis by iodine in obese strain chrickens[J]. Endocinology, 1995,136: 5054-5060.
  • 7Weetman AP, McGregor AM. Autoimmune thyroid disease: further development in our understanding[J]. Endoer Rev, 1994, 15: 788-830.
  • 8Filiphick P, Saito H, Katsuki H, et al. 3,5-3'-L-triiodothyronine promotes survival anf axon elongation of embryonic rat mptal neurons[J]. Brain Res, 1994,647: 148-152.
  • 9Mutaku JF,Poma JF, Many MC, et al. Cell necrosis and apoptosis are differentially regulated during goitre development and iodine-induced involution[J]. J Endocrinol, 2002, 172: 375-386.
  • 10FAO/WHO. Trace elements in human nutrition and health [M].Geneva: WHO, 1996: 49.

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