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青光眼视网膜神经节细胞的研究进展 被引量：2

A review of research advances on retinal ganglion cells in glaucoma
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摘要 对青光眼造成视网膜神经节细胞损害的机制、物质基础和防治原则等方面的文献进行了总结。 Literature on retinal ganglion cells was reviewed in the report, and including optic ganglion cells damage and its mechanism, vital materiality factors, protection from optic nerve and principle treated and otherwise
作者 李建超 彭清华
机构地区 湖南中医学院
出处 《中国中医眼科杂志》 2001年第2期119-123,共5页 China Journal of Chinese Ophthalmology
关键词 基因调控 青光眼 视网膜神经节细胞 glaucoma retinal ganglion cells,RGCs
分类号 R775.02 [医药卫生—眼科]
  • 相关文献

参考文献2

二级参考文献3

  • 1Michel Weber,Nicole Bonaventure,José A. Sahel. Protective role of excitatory amino acid antagonists in experimental retinal ischemia[J] 1995,Graefe’s Archive for Clinical and Experimental Ophthalmology(6):360～365
  • 2Yoshiaki Kitazawa,Hisayuki Shirai,Fu Jin Go. The effect of Ca2+-antagonist on visual field in low-tension glaucoma[J] 1989,Graefe’s Archive for Clinical and Experimental Ophthalmology(5):408～412
  • 3王守境,特检科,康维强.二维超声和脉冲波多普勒对青光眼眼血流动力学研究[J].眼科新进展,1992,12(1):9-11. 被引量：18

共引文献18

同被引文献38

  • 1卢艳,盛树力,吴航.大鼠视网膜神经网膜上神经营养因子的表达[J].眼科新进展,2002,22(1):21-22. 被引量：8
  • 2[1]Palmowski AM, Sutler EE, Bearse MA,et al.Mapping of retinal function in diabetic retinopathy using the multifocal electroretinogram.Invest Ophthalnol Vis Sci,1997,38(11):2586～2596
  • 3[3]Barber AT, Lieth E, Khin SA, et al. Neural apoptosis in the retina during experimental and human diabetes early onset and effect of insulin. Clin Invest,1998, 102(4): 783～719
  • 4[4]Moore P, ELsherbeny A, Room P, et al. Apoptosis cell death in the mouse retinal ganglion cell layer is induced in vivo by the excitatory amino acid homocysteine. Exp Eye Res, 2001,73(1): 45～57
  • 5[6]Brubaker RF. Delayed functional loss glaucoma L11 Edward Jackson memorial lecture[J]. Am J Ophthalmol, 1996, 12(3):473～483
  • 6[7]Fletcher EL, Kalloniatis M. Localization of amino acid neurotra-nsmitters during postnatal development of the rat retina . J Comp Neurol, 1997,380(3):449～471
  • 7[8]Rocha M, Martins Rap, Linden R. Activation of NMDA receptors protects against glutamate neurotoxicity in the neurotrophins. Brain Res,1999,827(1):79～92
  • 8[9]Podesta F, Romeo G, Liu WH, et al. Bax is increased in the retina of diabetic subjects and is associated with pericyte apoptosis in vivi and in vitro . Am J Pathol,2000, 156(3):1025～1032
  • 9[10]Sparapani M, Dall'Olio R, Gandolfi O ,et al. Neurotoxi city of polyamines and harmacological neuroprotection in cultures of rat cerebellar granule cells. Exp Neurol, 1997,148(1): 157～166
  • 10[20]Chierze S, Fehsel K, Kroncke K.D, et al. Protection of retinal ganglion cells and preservation of function after optic nerve lesion in bcl-2 transgenic mice. Visison Res, 1998,38:1547～1550

引证文献2

二级引证文献2

中国中医眼科杂志
2001年 第2期

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