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糖尿病血脂异常的发生机制 被引量:28

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作者 邓华聪 孙辽
出处 《辽宁实用糖尿病杂志》 2001年第1期5-7,共3页 Liaoning Journal of Practical Diabetology
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  • 1Bonnet F, Looper ME. Potential influence of lipid in diabetic nephropathy: insights from experimental data and clinical studid. Diabetes Metab 2000, 26(4) :254.
  • 2James RW, Boemil M, Sirolla C. et al. Lipoprotein(α) and vascular disease in diabetic patients. Diabetologia 1995,38:711.
  • 3Luiken JJ, Glatz JF Bonen A. Fatty acid transport proteins facilitate fatty acid uptake in skeletal muscle. Can J Appl Physiol2000, 25(5) :333.
  • 4Georgopoulos A, Aras D, Tsai MY, et al. Codon-54 polymorphism of the fatty acid-bilding protein-2 gene is associated with elevation of fasting and postprandial triglyceride in type-2 diabetes. J Clin Endocrinol Metab 2000, 85(9) :3155.
  • 5Bernard S, Touzet S, Personne I, et al. Association between microsomal transfer protein gene polymorphism and the biological feature of liver steslosis in patients with type-2 diabetes.Diabetologia 2000,43(8) :995.
  • 6Rangenathen G, Lio C, Kern RA. The translational regulation of lipoprotein lipese in diabeticrats involves the 3 untranslated region of the LPL mRNA. J Biol Chem 2000, 6 (record as suppled by publisher).
  • 7Memon RA, Tecott LH, Nonogaki K, et al. Up-regulation of peroxisome preliferator-activated receptors (PPAR-aplha) and PPAR-gamma messeager ribonucleic acid epression n the liver in murine obesity: triglitazone induces expression of PPAR-gammaresponsive adipose tissue-specific genes in the liver of obese diabetic mice. Endocrinology 2000,141(11) :4021.

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