摘要
目的 探讨病理性联带运动的临床表现和产生机制。方法 通过瞬目反射临床和实验测试重现病理性联带运动 ,应用扫描电镜观察神经纤维和肌纤维的神经—肌结合部的变化。结果 3 2例病理性联带运动全部为失神经支配Ⅲ度和重度面神经麻痹。 81% (2 8/3 2 )病例和 89%豚鼠瞬目反射测试在刺激侧同时记录到眼轮匝肌和口轮匝肌反应波形。结论 病理性联带运动是重度面神经麻痹的标志 。
Objective The clinical manifestations and mechanism of pathological synkinesia were studied. Methods Recurrent pathological synkinesia was measured by clinical and experimental nictitation reflex. The changes of nerve muscle junctions were observed via scanning electron microscope. Results All the 32 cases of pathological synkinesia were denervation of degree Ⅲ and severe facial palsy. Reaction undulatory forms of musculus orbicularis oculi and musculus orbicularis oris recorded simultaneously at the stimulated side in trigemial facial nerve reflex test accounted for 81%(28/32) patients and 89% guinea pigs. Conclusion Pathological synkinesia was the marker of severe facial palsy and was related with over error in regeneration of nerve.
出处
《中华物理医学与康复杂志》
CAS
CSCD
北大核心
2001年第1期38-40,共3页
Chinese Journal of Physical Medicine and Rehabilitation
