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中枢神经元钙激活钾通道的研究进展与缺血性脑损伤 被引量:1

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出处 《四川医学》 CAS 2001年第3期305-306,共2页 Sichuan Medical Journal
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  • 1[1]Stocker M,et al. An apamin-sensitive Ca2+-activated K+ current in hippocampal pyramidal neurons. Proc Nad Acad Sci, USA, 1999,96:4662
  • 2[2]Julie TC,et al. Cloning expression and distribution of functionally distinct Ca2+-activated K+ channel isoforms from human brain.Neuron, 1994,13:1315
  • 3[3]Nelson MT,et al. Physiologic roles and properties of potassium channels in arterial smooth muscle. Am J Physiol. 1995,268 :C799
  • 4[4]Bargas J,et al. Ca2+-activated outward currents in neostrital neurons. Neuroscience, 1999,88:479
  • 5[5]Neil VM,et al. Selective activation of Ca2+-activated K+ channels by co-localized Cag+ channels in hippocampal neurons. Nature,1998,395:900
  • 6[6]Sah P,et al. Channels underlying the slow afterhyperpolarization in hippocampal pyramidal neurons: neurotransmitters modulate the open probability. Neuron, 1995,15:435
  • 7[7]Pedarzani P,et al. Evidence that Ca2+/calmodulin-dependent protein kinase mediates the modulation of the Ca2+-dependent K+current,IHP,by acetylcholine,but not by glutamate,in hippcampal neurons. Eur J Physiol,1996. 431:723
  • 8[8]Pedarzani P,et al. Interaction between a and β -adrenergic receptor agonists modulating the slow Ca2+ activated K-+ current IAHp inhippocampal neurons. EJN, 1996,8 :2098
  • 9[9]Gold MS,et al. Role of a Ca2+-dependent slow afterhyperpolarization in prostaglandin E2-induced sensitization of cultured rat sensory neurons. Neurosci Lett. 1996.205:161
  • 10[10]Suroneier DJ,et al. Modulatin of cacium currents by a D1 dopaminergic protein kinase phosphate cascade in rat neostriata neurons.Neuron,1995,14:385

同被引文献25

  • 1张海霞,李正斌,王晓良.大脑中动脉栓塞模型大鼠的中枢电压依赖性钾通道mRNA表达的改变[J].药学学报,2006,41(4):328-332. 被引量:6
  • 2Kunz A, Dirnagl U, Mergenthaler P. Acute pathophysiological processes after ischaemic and traumatic brain injury[ J ]. Best Pract Res Clin Anaesthesiol, 2010,24 (4) : 495 -509.
  • 3Dirnagl U, ladecola C, Moskowitz MA. Pathobiology of ischaemic stroke: anintegrated view [J]. Trends Neurosci, 1999,22(9) :391-397.
  • 4Yu SP, Yeh C, Strasser U, et al. NMDA receptor-mediated K + effluxand neuronal apoptosis [J]. Science, 1999, 284(5412) :336-339.
  • 5Yu SP, Yeh CH, Sensi SL, et al. Mediation of neuronal apoptosis by enhancement of outwardpotassium current [J]. Science, 1997 ,278(5335) :114-117.
  • 6Shieh CC, Coghlan M, Sullivan JP, et al. Potassium channels: molecular defects, diseases, and therapeutic opportunities [J]. Pharmacol Rev, 2000, 52 (4): 557-594.
  • 7Huang H, Gao TM, Gong L, et al. Potassium channel blocker TEAprevents CAl hippocampal injury following transient forebrain ischemia in adultrats [J]. N eurosci Lett,2001 ,305(2) :83-86.
  • 8Chi S, Cai W, Liu P, et al. Baifuzireduces transient ischemic brain damage through an interaction with the STREXdomain of BKCa channels [J]. Cell Death Dis, 2010,1(13) :1-11.
  • 9Zetterstrom TS, Vaughan-Jones RD, Grahame-Smith DG. A short period of hypoxia produces a rapid and transient rise in [K+] e in rat hippocampus in vivo which is inhibited by certain K ( + ) -channel blocking agents [J] . Neuroscience, 1995,67 (4) :815-821.
  • 10Dos Santos P, Kowaltowski AJ, Laclau MN, et al. Mechanisms by which opening the mitochondrialATP- sensitive K( + ) channel protects the ischemic heart [ J ]. Am J Physiol Heart Circ Physiol,2002,283 (1) :H284- H 295.

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