雷米普利减轻高血压病患者左室重量的效果优于氨酰心安——雷米普利心肌保护作用评价
摘要
高血压病患者发生左室肥厚的主要机制之一是适应异常负荷情况的反应,即心脏重量的增加是缘于适应过重的心脏后负荷的结果。高血压初期阶段左室心肌肥厚是一种有用的代偿过程,在某种程度上肥厚僵硬的心肌有防止心空腔进一步扩大的作用。据统计,临床仅有1/3高血压病患者合并左心室肥厚。伴随左心室肥厚的发生,一些临床疾病也逐渐明显的表现出来。如高血压性心力衰竭、缺血性心脏病、心律失常、甚至出现碎死等。Fram-ingham研究结果认为左室肥厚是一个不详的预兆。
出处
《中国医药导刊》
2000年第2期14-17,共4页
Chinese Journal of Medicinal Guide
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