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乙型肝炎病毒持续感染的可能机制 被引量:5

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作者 季伟 王慧芬
机构地区 解放军
出处 《临床肝胆病杂志》 CAS 北大核心 2000年第4期201-202,共2页 Journal of Clinical Hepatology
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参考文献7

  • 1Chisari FV and Ferrari C.Hepatitie B virus immunopathogenesis[J] .Annu Rev Immunol, 1995,13 ; 29.
  • 2Guidotti LG, Rochford R,Chung J, et al. Viral clearance without destruction of infected cells during acute HBV infection[J] .Science,1999, 284(5415):825.
  • 3Chisari FV. Cytotonic T cells and viral hepatitis[J] .J Clin Inveat, 1997,99 : 1742.
  • 4Kato J,Hasegawa K,Toril N, et al.A molecular analysis of viral persistece in surfaoe antigen-negative chronic hepatitis B[J]. Hepstology,1996,23:389.
  • 5Lok ASF, Akarca U, Greene S. Mutations in the precore region of hepatitis B virus serve to enhance the stability of the secondary structure of the pregenome encapsidation signal[J] .Proc Natl Acad Sci USA,1994,91;4077.
  • 6Bertoletti A,Costanzo A, Chisari FV,et al. Cytotoxic T lymphocyte response to a wild-type hepatitis B virus epitope in patients chroaically infected by viruses carrying substitutions withis the epitope[J] .J Exp Med,1994,180:933. M~I,1994,180:933
  • 7Sette A,Alexander J,Ruppert J,et al.antigen analogs/MHC complexes as specific T cell receptor antagonists[J] .Amu,Rev Immunol. 1994,12:413.

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