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再灌注损伤挽救激酶和线粒体膜通透性转换在肝缺血后处理中的作用 被引量:2

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摘要 缺血后处理(IPO)能减轻肝脏缺血再灌注损伤。我们通过观察缺血后处理对磷脂酰肌醇-3激酶(P13K)、细胞外信号调节激酶1/2(ERK1/2)和线粒体膜通透性转换(MPT)的影响,探讨肝脏IPO的作用机制。
出处 《中华实验外科杂志》 CAS CSCD 北大核心 2012年第5期975-976,共2页 Chinese Journal of Experimental Surgery
基金 陕西省科技攻关计划资助项目(2008K14-02)
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