期刊文献+

柴胡止血液治疗宫内节育器所致子宫出血的临床研究——附:60例病例报告 被引量:16

Clinical Observation on Treatment of Endometriosis by Tonifying Qi and Promoting Blood Circulation to Remove Stasis and Purgation Principle
暂未订购
导出
摘要 目的 :观察柴胡止血液与消炎痛等抗前列腺素合成剂对IUD所致子宫异常出血的疗效。方法 :采用柴胡止血液治疗组与消炎痛等对照组对 60例IUD所致子宫异常出血患者进行临床治疗。结果 :治疗组临床治愈 8例 ,显效 16例 ,无效 6例 ,总有效率为 80 .0 0 % ;对照组临床治愈 2例 ,显效 18例 ,无效 10例 ,总有效率为 66.67% ,两组比较无统计学意义 (P >0 0 5 )。柴胡止血液在减少置器者月经血量及经期时间方面 ,与治疗前比较均有高度统计学意义 (P <0 0 1) ,在减少经期时间上与对照组比较有统计学意义 (P <0 0 5 ) ,在减少月经血量方面与对照组比较无统计学意义 (P >0 0 5 )。结论 :柴胡止血液可能通过调整了前列腺素的含量以及修复子宫内膜的作用、抗炎和抗菌等协同作用 ,在治疗IUD所致子宫异常出血方面具有较好疗效。 To explore the therapeutic mechanism of endometriosis by the treatment of Yiqi Huoxue Huayu Tongfu Principle (YQHXHYTFP, tonifying Qi and promoting blood circulation to remove stasis and purgation therapy) with Neiyi 1 + pill. Methods: Forty five cases with endometriosis were divided randomly into TCM group (30 cases, treated with Neiyi 1 + pill) and the control group (15 cases, treated with tamoxifen). The activity of natural killer cell of peripheral blood, T lymphocyte subsets, secreting interleukin Ⅱ and the EmAb were determined. Results: Endometriosis is associated with immunologic disturbance. After treatment, the activity of NK cells and the count of T suppressor cells were significantly increased (P<0 01), the count of T helper cells, the secretion of interleukin Ⅱ and TH/TS ratio were significantly reduced (P<0 01). Over 50% of the cases whose EmAb was positive eventually turned to negative. The effective rate of this therapy was 90%. There was no significant difference between TCM group and the control group. Conclusion: YQHXHYTF therapy could modulate the immunologic disturbance in women with endometriosis, improve the clinical symptoms and signs obviously. It is a safe and effective therapy.
出处 《成都中医药大学学报》 1999年第4期6-8,共3页 Journal of Chengdu University of Traditional Chinese Medicine
基金 四川省教委科研基金
关键词 柴胡止血液 节育器 子宫异常出血 中医药疗法 endometriosis Yiqi Huoxue Huayu Tongfu Principle immunoregulation tamoxifen
  • 相关文献

参考文献6

二级参考文献10

共引文献129

同被引文献129

引证文献16

二级引证文献30

相关作者

内容加载中请稍等...

相关机构

内容加载中请稍等...

相关主题

内容加载中请稍等...

浏览历史

内容加载中请稍等...
;
使用帮助 返回顶部