3Bianchi ME,Manfredi AA.High mobility group box 1(HMGB1) protein at the cross roads between innate and adaptive immunity[J].Immunol Rev,2007;220(1):35-46.
7Kokkola R,Andersson A,Mullins G,et al.RAGE is the major receptor for the proinflammatory activity of HMGB1 in rodent macrophages[J].Scand J Immunol,2005;61(1):1.
1Wang H,Bloom O,Zhang M,et al. HMG-1 as a late mediator of endotoxin lethality in mice. Science,1999,285:248-251.
2Huttunen HJ,Rauvala H. Amphoterin as an extracellular regulator of cell motility: from discovery to disease. J Intern Med, 2004,255 : 351-366.
3Chen G, Li J, Qiang X, et al. Suppression of HMGB1 release by stearoyl lysophosphatidylcholine:an additional mechanism for its therapeutic effects in experimental sepsis. J Lipid Res, 2005,46 : 623-627.
4Kalinina N, Agrotis A, Antropova Y, et al. Increased expres sion of the DNA-binding cytokine HMGB1 in human athero sclerotic lesions: role of activated macrophages and cyto kines. Arterioscler Thromb Vasc Biol, 2004,24 : 2320-2325.
5Inoue K, Kawahara K, Biswas KK, et al. HMGB1 expression by activated vascular smooth muscle cells in advanced human atherosclerosis plaques. Cardiovasc Pathol, 2007,16 : 136-143.
6Abeyama K,Stern DM, Ito Y, et al. The N-terminal domain of thrombomodulin sequesters high-mobility groulp-B1 protein, a novel antiinflammatory mechanism. J Clin Invest, 2005,115 : 1267-1274.
7Porto A,Palumbo R,Pieroni M, et al. Smooth muscle ceils in human atherosclerotic plaques secrete and proliferate in response to high mobility group box 1 protein. FASEB J ,2006, 20:2565-2566.
8Basra G. Receptor for advanced glycation endproducts and atherosclerosis: From basic mechanisms to clinical implications. Atherosclerosis, 2008,196 : 9-21.
9Amandine J, Sylvain T,Brigitte J, et al. Activation of sPLA2- IIA and PGE2 production by high mobility group protein B1 in vascular smooth muscle cells sensitized by IL-lbeta. FASEB J,2006,20:1727-1729.
10Bradshaw G, Gutierrez A, Miyake JH, et al. Facilitated replacement of Kupffer cells expressing a paraoxonase-1 transgene is essential for ameliorating atherosclerosis in mice. Proe Natl Acad Sci USA, 2005,102:11029-11034.
