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晚期糖基化终末产物受体与阿尔茨海默病 被引量：11

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作者 丛潇 王友政 张文生
机构地区 北京师范大学资源学院资源药物与中药资源研究所教育部资源药物工程研究中心
出处 《中国老年学杂志》 CAS CSCD 北大核心 2010年第12期1759-1761,共3页 Chinese Journal of Gerontology
基金 北京市教育委员会新医药学科群项目(XK100270569) 北京市教育委员会产学研合作项目(105805)
关键词 晚期糖基化终产物受体 阿尔茨海默病 β样淀粉蛋白
分类号 R74 [医药卫生—神经病学与精神病学]
  • 相关文献

参考文献26

  • 1Akiyama H,Barger S,Barnum S,et al.Inflammation and Alzheimer′s disease[J].Neurobiol Aging,2000;21(3):383-421.
  • 2David D,Shelley JA.Neurobiology of Alzheimer′s disease[M].3rd ed.Oxford University Press,2007:227-33.
  • 3D′Hooge R,Nagels G,Westland CE.Spatial learning deficit in mice expressing human 751-amino acid beta-amyloid precursor protein[J].Neuroreport,1996;7(15-17):2807-11.
  • 4Ingelsson M,Fukumoto H,Newell KL,et al.Early Aβ accumulation and progressive synaptic loss,gliosis,and tangle formation in AD brain[J].Neurology,2004;62(6):925-31.
  • 5Chen X,Walker DG,Schmiolt AM,et al.RAGE:a potential target for Aβ-mediated cellular perturbation in Alzheimer′s disease[J].Curr Molecul Med,2007;7(8):735-42.
  • 6Schmidt AM,Yan SD,Yan SF,et al.The multiligand receptor RAGE as a progression factor amplifying immune and inflammatory responses[J].J Clin Invest,2001;108:949-55.
  • 7王瑞良,苏青.RAGE及其配体的临床意义研究进展[J].国际内分泌代谢杂志,2006,26(3):160-162. 被引量：23
  • 8Chaneya MO,Stine WB,Kokjohn TA,et al.RAGE and amyloid-beta interactions:Atomic force microscopy and molecular modeling[J].Biochim Biophys Acta,2005;174(1-2):199-205.
  • 9陈媛,周玫.自由基炎症与衰老性疾病[M].北京:科学出版社,2007:342-71.
  • 10赵善超,刘靖华,李志杰,秦清和,邓鹏,侯凡凡,姜勇.人晚期糖基化终产物受体胞质内段融合蛋白表达载体的构建与表达[J].第一军医大学学报,2003,23(12):1314-1316. 被引量：7

二级参考文献24

  • 1[3]Neeper M, Schmidt AM, Brett J, et al. Cloning and expression of a cell surface receptor for advanced glycosylation endproducts of protein[J ]. J Biol Chem, 1992, 267(21): 14998-5004.
  • 2[4]Vlassara H, Bucala R, Striker L, et al. Pathogenic effects of advanced glycosylation:biochemical, biologic, and clinical implications for diabetes and aging[J]. Lab Invest, 1994, 70(2): 138-51.
  • 3Forbes JM, Thallas V, Thomas MC, et al. The breakdown of preexisting advanced glycation end products is associated with reduced renal fibrosis in experimental diabetes. FASEB J, 2003,17 : 1762-1764.
  • 4Candido R, Forbes JM, Thomas MC, et al. A breaker of advanced glycation end products attenuates diabetes-induced myocardial structural changes: Circ Res, 2003,92:785-792.
  • 5Haslbeck KM, Schleieher E, Bierhaus A, et al. The AGEVRAGE/NF-kappaB pathway may contribute to the pathogenesis of polyneuropathy in impaired glucose tolerance (IGT). Exp Clin Endocrinol Diabetes,2005,113 : 288-291.
  • 6Prevost G, Fajardy I, Besmond C, et al. Polymorphisms of the neceptor of advanced glycation end products (RAGE) and the development of nephropathy in type 1 diabetic patients. Diabetes Metab, 2005, 31 : 35-39.
  • 7Kankova K, Stejskalova A, Hertlova M, et al. Haplotype analysis of the RAGE gene: identification of a haplotype marker for diabetic nephropathy in type 2 diabetes mellitus. Nephrol Dial Transplant, 2005,20: 1093-1102.
  • 8Yamamoto Y, Kato I, Doi T, et al. Development and prevention of advanced diabetic nephropathy in RAGE-overexpressing mice. J Clin Invest,2001,108:261-268.
  • 9Wendt TM, Tanji N, Guo J, et al. RAGE drives the development of glomerulosclerosis and implicates podocyte activation in the pathogenesis of diabetic nephropathy. Am J Pathol, 2003,162:1123-1137.
  • 10Yan SF, Ramasamy R, Naka Y, et al. Glycation,inflammation,and RAGE:a scaffold for the macrovascular complications of diabetes and beyond. Circ Res, 2003,93 : 1159-1169.

共引文献28

同被引文献218

引证文献11

二级引证文献78

中国老年学杂志
2010年 第12期

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